pubmed-article:15282385 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C0004096 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C0001927 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C0060657 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C0073992 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C1704410 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C1152728 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C1709305 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C0205272 | lld:lifeskim |
pubmed-article:15282385 | lifeskim:mentions | umls-concept:C2828360 | lld:lifeskim |
pubmed-article:15282385 | pubmed:issue | 8 | lld:pubmed |
pubmed-article:15282385 | pubmed:dateCreated | 2004-7-29 | lld:pubmed |
pubmed-article:15282385 | pubmed:abstractText | The development of tolerance following the use of long acting beta(2) agonists in asthmatic patients with either the homozygous arginine (Arg-16) or glycine (Gly-16) genotypes is poorly documented, especially in relation to the acute reliever response to salbutamol in constricted airways. A study was undertaken to evaluate the Arg-16 and Gly-16 genotypes for the acute salbutamol response following methacholine bronchial challenge between the first and last doses of formoterol (FM) and salmeterol (SM) combination inhalers. | lld:pubmed |
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pubmed-article:15282385 | pubmed:language | eng | lld:pubmed |
pubmed-article:15282385 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15282385 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:15282385 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:15282385 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15282385 | pubmed:month | Aug | lld:pubmed |
pubmed-article:15282385 | pubmed:issn | 0040-6376 | lld:pubmed |
pubmed-article:15282385 | pubmed:author | pubmed-author:JacksonC MCM | lld:pubmed |
pubmed-article:15282385 | pubmed:author | pubmed-author:BatesC ECE | lld:pubmed |
pubmed-article:15282385 | pubmed:author | pubmed-author:LipworthB JBJ | lld:pubmed |
pubmed-article:15282385 | pubmed:author | pubmed-author:LeeD K CDK | lld:pubmed |
pubmed-article:15282385 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15282385 | pubmed:volume | 59 | lld:pubmed |
pubmed-article:15282385 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15282385 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15282385 | pubmed:pagination | 662-7 | lld:pubmed |
pubmed-article:15282385 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:15282385 | pubmed:year | 2004 | lld:pubmed |
pubmed-article:15282385 | pubmed:articleTitle | Cross tolerance to salbutamol occurs independently of beta2 adrenoceptor genotype-16 in asthmatic patients receiving regular formoterol or salmeterol. | lld:pubmed |
pubmed-article:15282385 | pubmed:affiliation | Asthma and Allergy Research Group, Ninewells Hospital and Medical School, University of Dundee, Dundee DD1 9SY, UK. | lld:pubmed |
pubmed-article:15282385 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15282385 | pubmed:publicationType | Clinical Trial | lld:pubmed |
pubmed-article:15282385 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
pubmed-article:15282385 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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