| pubmed-article:15269141 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15269141 | lifeskim:mentions | umls-concept:C0020971 | lld:lifeskim |
| pubmed-article:15269141 | lifeskim:mentions | umls-concept:C0027651 | lld:lifeskim |
| pubmed-article:15269141 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:15269141 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:15269141 | lifeskim:mentions | umls-concept:C0087071 | lld:lifeskim |
| pubmed-article:15269141 | lifeskim:mentions | umls-concept:C0030956 | lld:lifeskim |
| pubmed-article:15269141 | lifeskim:mentions | umls-concept:C1705316 | lld:lifeskim |
| pubmed-article:15269141 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:15269141 | pubmed:issue | 14 | lld:pubmed |
| pubmed-article:15269141 | pubmed:dateCreated | 2004-7-22 | lld:pubmed |
| pubmed-article:15269141 | pubmed:abstractText | Telomerase is an attractive target antigen for cancer immunotherapies because it is expressed in >85% of human tumors but is rarely found in normal tissues. A HLA-A*0201-restricted T-cell epitope was previously identified within telomerase reverse transcriptase hTERT:540-548. This peptide was reported to induce CTL that recognized tumor cells and transfectants that endogenously expressed telomerase. Therefore, we initiated a clinical protocol to evaluate the therapeutic and immunological efficacy of this peptide. | lld:pubmed |
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| pubmed-article:15269141 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15269141 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15269141 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15269141 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:15269141 | pubmed:issn | 1078-0432 | lld:pubmed |
| pubmed-article:15269141 | pubmed:author | pubmed-author:LiYongY | lld:pubmed |
| pubmed-article:15269141 | pubmed:author | pubmed-author:IgarashiTakeh... | lld:pubmed |
| pubmed-article:15269141 | pubmed:author | pubmed-author:RosenbergStev... | lld:pubmed |
| pubmed-article:15269141 | pubmed:author | pubmed-author:RileyJohn PJP | lld:pubmed |
| pubmed-article:15269141 | pubmed:author | pubmed-author:ParkhurstMari... | lld:pubmed |
| pubmed-article:15269141 | pubmed:author | pubmed-author:RobbinsPaul... | lld:pubmed |
| pubmed-article:15269141 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15269141 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:15269141 | pubmed:volume | 10 | lld:pubmed |
| pubmed-article:15269141 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15269141 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15269141 | pubmed:pagination | 4688-98 | lld:pubmed |
| pubmed-article:15269141 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:15269141 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15269141 | pubmed:articleTitle | Immunization of patients with the hTERT:540-548 peptide induces peptide-reactive T lymphocytes that do not recognize tumors endogenously expressing telomerase. | lld:pubmed |
| pubmed-article:15269141 | pubmed:affiliation | National Cancer Institute, Surgery Branch and National Heart, Lung, and Blood Institute, Hematology Branch, NIH, Bethesda, Maryland 20892-1502, USA. Maria_Parkhurst@nih.gov | lld:pubmed |
| pubmed-article:15269141 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15269141 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:15269141 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
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