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| pubmed-article:15213174 | lifeskim:mentions | umls-concept:C0006852 | lld:lifeskim |
| pubmed-article:15213174 | lifeskim:mentions | umls-concept:C0012655 | lld:lifeskim |
| pubmed-article:15213174 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:15213174 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:15213174 | lifeskim:mentions | umls-concept:C1414863 | lld:lifeskim |
| pubmed-article:15213174 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:15213174 | lifeskim:mentions | umls-concept:C1517004 | lld:lifeskim |
| pubmed-article:15213174 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:15213174 | pubmed:dateCreated | 2004-6-23 | lld:pubmed |
| pubmed-article:15213174 | pubmed:abstractText | Fut2-LacZ-null mice, which are a model of the human ABO and Lewis nonsecretor group, display increased susceptibility to experimental yeast vaginitis, indicating a role for alpha(1,2)fucosylated cervical glycans in mucosal defense. However, the lack of significant effect of competitive inhibition by exogenous neoglycoproteins in this study emphasizes the complexity of Candida-epithelial cell adhesion events. | lld:pubmed |
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| pubmed-article:15213174 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15213174 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15213174 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15213174 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:15213174 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:15213174 | pubmed:author | pubmed-author:DominoSteven... | lld:pubmed |
| pubmed-article:15213174 | pubmed:author | pubmed-author:HurdElizabeth... | lld:pubmed |
| pubmed-article:15213174 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15213174 | pubmed:volume | 72 | lld:pubmed |
| pubmed-article:15213174 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15213174 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15213174 | pubmed:pagination | 4279-81 | lld:pubmed |
| pubmed-article:15213174 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:15213174 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15213174 | pubmed:articleTitle | Increased susceptibility of secretor factor gene Fut2-null mice to experimental vaginal candidiasis. | lld:pubmed |
| pubmed-article:15213174 | pubmed:affiliation | Department of Obstetrics and Gynecology, 6428 Medical Science I, Box 0617, University of Michigan, Ann Arbor, MI 48109-0617, USA. | lld:pubmed |
| pubmed-article:15213174 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15213174 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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