| pubmed-article:15184287 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:15184287 | lifeskim:mentions | umls-concept:C0072980 | lld:lifeskim |
| pubmed-article:15184287 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:15184287 | lifeskim:mentions | umls-concept:C0033095 | lld:lifeskim |
| pubmed-article:15184287 | lifeskim:mentions | umls-concept:C1383860 | lld:lifeskim |
| pubmed-article:15184287 | lifeskim:mentions | umls-concept:C1710082 | lld:lifeskim |
| pubmed-article:15184287 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:15184287 | lifeskim:mentions | umls-concept:C0443211 | lld:lifeskim |
| pubmed-article:15184287 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:15184287 | pubmed:issue | 24 | lld:pubmed |
| pubmed-article:15184287 | pubmed:dateCreated | 2004-6-22 | lld:pubmed |
| pubmed-article:15184287 | pubmed:abstractText | Rapamycin is a specific inhibitor of the mammalian target of rapamycin (mTOR). We recently reported that administration of rapamycin before exposure to ascending aortic constriction significantly attenuated the load-induced increase in heart weight by approximately 70%. | lld:pubmed |
| pubmed-article:15184287 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15184287 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15184287 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:15184287 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15184287 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:15184287 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:15184287 | pubmed:author | pubmed-author:ZhangLiL | lld:pubmed |
| pubmed-article:15184287 | pubmed:author | pubmed-author:IzumoSeigoS | lld:pubmed |
| pubmed-article:15184287 | pubmed:author | pubmed-author:McMullenJulie... | lld:pubmed |
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| pubmed-article:15184287 | pubmed:author | pubmed-author:ShioiTetsuoT | lld:pubmed |
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| pubmed-article:15184287 | pubmed:author | pubmed-author:DorfmanAdam... | lld:pubmed |
| pubmed-article:15184287 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:15184287 | pubmed:day | 22 | lld:pubmed |
| pubmed-article:15184287 | pubmed:volume | 109 | lld:pubmed |
| pubmed-article:15184287 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15184287 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15184287 | pubmed:pagination | 3050-5 | lld:pubmed |
| pubmed-article:15184287 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:15184287 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15184287 | pubmed:articleTitle | Inhibition of mTOR signaling with rapamycin regresses established cardiac hypertrophy induced by pressure overload. | lld:pubmed |
| pubmed-article:15184287 | pubmed:affiliation | Cardiovascular Division, Department of Medicine, Beth Israel Deaconess Medical Center, 330 Brookline Ave, Boston, Mass 02215, USA. jmcmulle@bidmc.harvard.edu | lld:pubmed |
| pubmed-article:15184287 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15184287 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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