| pubmed-article:1516259 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0032961 | lld:lifeskim |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0003864 | lld:lifeskim |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0020852 | lld:lifeskim |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0001721 | lld:lifeskim |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0439793 | lld:lifeskim |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0449438 | lld:lifeskim |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0017982 | lld:lifeskim |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0021149 | lld:lifeskim |
| pubmed-article:1516259 | lifeskim:mentions | umls-concept:C0081484 | lld:lifeskim |
| pubmed-article:1516259 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:1516259 | pubmed:dateCreated | 1992-10-7 | lld:pubmed |
| pubmed-article:1516259 | pubmed:abstractText | The effect of pregnancy on the incidence and severity of pristane-induced arthritis was examined along with the glycosylation status of IgG during the ante-natal and post-partum periods. It was found that pristane-induced arthritis is prevented by pregnancy. In addition, the levels of agalactosyl IgG fall during pregnancy but rise to greater than normal within a few days of parturition, before resetting towards the norm shortly afterwards. Interestingly, the level of agalactosyl IgG correlates with the severity of arthritis. As previously reported IL-6 may be an important factor, not necessarily the only one, in the production of agalactosyl IgG. Here it is clearly demonstrated that the kinetics of IL-6 activity post-pristane injection parallels the kinetics of agalactosyl IgG production. In addition, the overshoot in agalactosyl IgG levels immediately post-partum coincides with a burst in IL-6 activity. It is considered that these changes in IgG glycoform levels, or the factors which control them, may be related to the mechanisms underlying prevention/remission of arthritis during pregnancy. | lld:pubmed |
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| pubmed-article:1516259 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1516259 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1516259 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1516259 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1516259 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:1516259 | pubmed:issn | 0009-9104 | lld:pubmed |
| pubmed-article:1516259 | pubmed:author | pubmed-author:ElsonC JCJ | lld:pubmed |
| pubmed-article:1516259 | pubmed:author | pubmed-author:RookG AGA | lld:pubmed |
| pubmed-article:1516259 | pubmed:author | pubmed-author:ZhangY WYW | lld:pubmed |
| pubmed-article:1516259 | pubmed:author | pubmed-author:ThompsonS JSJ | lld:pubmed |
| pubmed-article:1516259 | pubmed:author | pubmed-author:HitsumotoYY | lld:pubmed |
| pubmed-article:1516259 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1516259 | pubmed:volume | 89 | lld:pubmed |
| pubmed-article:1516259 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1516259 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1516259 | pubmed:pagination | 434-8 | lld:pubmed |
| pubmed-article:1516259 | pubmed:dateRevised | 2010-9-7 | lld:pubmed |
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| pubmed-article:1516259 | pubmed:meshHeading | pubmed-meshheading:1516259-... | lld:pubmed |
| pubmed-article:1516259 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1516259 | pubmed:articleTitle | Agalactosyl IgG in pristane-induced arthritis. Pregnancy affects the incidence and severity of arthritis and the glycosylation status of IgG. | lld:pubmed |
| pubmed-article:1516259 | pubmed:affiliation | Department of Pathology and Microbiology, School of Medical Science, University of Bristol, UK. | lld:pubmed |
| pubmed-article:1516259 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1516259 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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