| pubmed-article:15135377 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15135377 | lifeskim:mentions | umls-concept:C0205112 | lld:lifeskim |
| pubmed-article:15135377 | lifeskim:mentions | umls-concept:C1801960 | lld:lifeskim |
| pubmed-article:15135377 | lifeskim:mentions | umls-concept:C1882727 | lld:lifeskim |
| pubmed-article:15135377 | lifeskim:mentions | umls-concept:C0018969 | lld:lifeskim |
| pubmed-article:15135377 | lifeskim:mentions | umls-concept:C0205409 | lld:lifeskim |
| pubmed-article:15135377 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:15135377 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
| pubmed-article:15135377 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:15135377 | pubmed:dateCreated | 2004-5-11 | lld:pubmed |
| pubmed-article:15135377 | pubmed:abstractText | Vasoconstriction in pulmonary ischemia-reperfusion injury may involve dysfunction of the physiologic vasodilation of pulmonary arteries. Little is known of the relative importance of heme oxygenase (HO)/carbon monoxide (CO)-dependent vs nitric oxide synthase (NOS)/nitric oxide (NO)-dependent vasodilation of the pulmonary vasculature. We evaluated the significance of HO function on basal pulmonary vascular resistance (PVR) and compared it with the function of NOS. | lld:pubmed |
| pubmed-article:15135377 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15135377 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15135377 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:15135377 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15135377 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15135377 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15135377 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15135377 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15135377 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15135377 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15135377 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15135377 | pubmed:month | May | lld:pubmed |
| pubmed-article:15135377 | pubmed:issn | 1053-2498 | lld:pubmed |
| pubmed-article:15135377 | pubmed:author | pubmed-author:NakamuraTakay... | lld:pubmed |
| pubmed-article:15135377 | pubmed:author | pubmed-author:MengerMichael... | lld:pubmed |
| pubmed-article:15135377 | pubmed:author | pubmed-author:WadaHiromiH | lld:pubmed |
| pubmed-article:15135377 | pubmed:author | pubmed-author:SchäfersHans-... | lld:pubmed |
| pubmed-article:15135377 | pubmed:author | pubmed-author:VollmarBrigit... | lld:pubmed |
| pubmed-article:15135377 | pubmed:author | pubmed-author:InuiKenjiK | lld:pubmed |
| pubmed-article:15135377 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15135377 | pubmed:volume | 23 | lld:pubmed |
| pubmed-article:15135377 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15135377 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15135377 | pubmed:pagination | 599-605 | lld:pubmed |
| pubmed-article:15135377 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:meshHeading | pubmed-meshheading:15135377... | lld:pubmed |
| pubmed-article:15135377 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15135377 | pubmed:articleTitle | Heme oxygenase does not contribute to control of basal vascular tone in isolated blood-perfused rat lung. | lld:pubmed |
| pubmed-article:15135377 | pubmed:affiliation | Institute for Clinical and Experimental Surgery, University of Saarland, Homburg/Saar, Germany. tnakamur@kuhp.kyoto-u.ac.ip | lld:pubmed |
| pubmed-article:15135377 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15135377 | pubmed:publicationType | In Vitro | lld:pubmed |
