| pubmed-article:15123368 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15123368 | lifeskim:mentions | umls-concept:C0019169 | lld:lifeskim |
| pubmed-article:15123368 | lifeskim:mentions | umls-concept:C0023895 | lld:lifeskim |
| pubmed-article:15123368 | lifeskim:mentions | umls-concept:C0205082 | lld:lifeskim |
| pubmed-article:15123368 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
| pubmed-article:15123368 | lifeskim:mentions | umls-concept:C1521991 | lld:lifeskim |
| pubmed-article:15123368 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:15123368 | lifeskim:mentions | umls-concept:C1292724 | lld:lifeskim |
| pubmed-article:15123368 | lifeskim:mentions | umls-concept:C0700164 | lld:lifeskim |
| pubmed-article:15123368 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:15123368 | pubmed:dateCreated | 2004-5-4 | lld:pubmed |
| pubmed-article:15123368 | pubmed:abstractText | PreS2-defective hepatitis B virus (HBV) variants may emerge during chronic HBV infection. These variants carry mutation(s) at the ATG-start-codon and/or in-frame deletion into the preS2 genomic region and are commonly detected by sequencing analyses. We evaluated the prevalence of these variants in a large series of chronic HBV infected patients through non-sequencing molecular approaches. | lld:pubmed |
| pubmed-article:15123368 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15123368 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15123368 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:15123368 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15123368 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15123368 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15123368 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15123368 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15123368 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:15123368 | pubmed:issn | 0168-8278 | lld:pubmed |
| pubmed-article:15123368 | pubmed:author | pubmed-author:SquadritoGiov... | lld:pubmed |
| pubmed-article:15123368 | pubmed:author | pubmed-author:PollicinoTere... | lld:pubmed |
| pubmed-article:15123368 | pubmed:author | pubmed-author:CacciolaIrene... | lld:pubmed |
| pubmed-article:15123368 | pubmed:author | pubmed-author:RaimondoGiova... | lld:pubmed |
| pubmed-article:15123368 | pubmed:author | pubmed-author:CaccamoGaiaG | lld:pubmed |
| pubmed-article:15123368 | pubmed:author | pubmed-author:CostantinoLuc... | lld:pubmed |
| pubmed-article:15123368 | pubmed:author | pubmed-author:BrancatelliSa... | lld:pubmed |
| pubmed-article:15123368 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15123368 | pubmed:volume | 40 | lld:pubmed |
| pubmed-article:15123368 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15123368 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15123368 | pubmed:pagination | 515-9 | lld:pubmed |
| pubmed-article:15123368 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:15123368 | pubmed:meshHeading | pubmed-meshheading:15123368... | lld:pubmed |
| pubmed-article:15123368 | pubmed:meshHeading | pubmed-meshheading:15123368... | lld:pubmed |
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| pubmed-article:15123368 | pubmed:meshHeading | pubmed-meshheading:15123368... | lld:pubmed |
| pubmed-article:15123368 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15123368 | pubmed:articleTitle | Non-sequencing molecular approaches to identify preS2-defective hepatitis B virus variants proved to be associated with severe liver diseases. | lld:pubmed |
| pubmed-article:15123368 | pubmed:affiliation | Unità di Epatologia Clinica e Biomolecolare, Dipartimento di Medicina Interna, Policlinico Universitario di Messina, 98124 Messina, Italy. raimondo@unime.it | lld:pubmed |
| pubmed-article:15123368 | pubmed:publicationType | Journal Article | lld:pubmed |
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