| pubmed-article:15106801 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15106801 | lifeskim:mentions | umls-concept:C0019425 | lld:lifeskim |
| pubmed-article:15106801 | lifeskim:mentions | umls-concept:C0001721 | lld:lifeskim |
| pubmed-article:15106801 | lifeskim:mentions | umls-concept:C0005823 | lld:lifeskim |
| pubmed-article:15106801 | lifeskim:mentions | umls-concept:C1413516 | lld:lifeskim |
| pubmed-article:15106801 | lifeskim:mentions | umls-concept:C0332453 | lld:lifeskim |
| pubmed-article:15106801 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:15106801 | pubmed:dateCreated | 2004-4-26 | lld:pubmed |
| pubmed-article:15106801 | pubmed:abstractText | It has been suspected that the mast cell chymase gene (CMA1) is important for the generation of angiotensin II and therefore might be associated with the pathogenesis of hypertension. | lld:pubmed |
| pubmed-article:15106801 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15106801 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15106801 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:15106801 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15106801 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15106801 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15106801 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15106801 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15106801 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:15106801 | pubmed:issn | 0263-6352 | lld:pubmed |
| pubmed-article:15106801 | pubmed:author | pubmed-author:IwaiNaoharuN | lld:pubmed |
| pubmed-article:15106801 | pubmed:author | pubmed-author:MannamiToshif... | lld:pubmed |
| pubmed-article:15106801 | pubmed:author | pubmed-author:YouW TWT | lld:pubmed |
| pubmed-article:15106801 | pubmed:author | pubmed-author:ShiojiKeisuke... | lld:pubmed |
| pubmed-article:15106801 | pubmed:author | pubmed-author:KokuboYoshihi... | lld:pubmed |
| pubmed-article:15106801 | pubmed:author | pubmed-author:InamotoNozomu... | lld:pubmed |
| pubmed-article:15106801 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15106801 | pubmed:volume | 22 | lld:pubmed |
| pubmed-article:15106801 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15106801 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15106801 | pubmed:pagination | 103-9 | lld:pubmed |
| pubmed-article:15106801 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:15106801 | pubmed:meshHeading | pubmed-meshheading:15106801... | lld:pubmed |
| pubmed-article:15106801 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15106801 | pubmed:articleTitle | Heterozygous disruption of CMA1 does not affect blood pressure. | lld:pubmed |
| pubmed-article:15106801 | pubmed:affiliation | National Cardiovascular Center, Suita, Osaka, Japan. | lld:pubmed |
| pubmed-article:15106801 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15106801 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:15106801 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:1215 | entrezgene:pubmed | pubmed-article:15106801 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:15106801 | lld:entrezgene |
