| pubmed-article:15084693 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15084693 | lifeskim:mentions | umls-concept:C0220615 | lld:lifeskim |
| pubmed-article:15084693 | lifeskim:mentions | umls-concept:C0445604 | lld:lifeskim |
| pubmed-article:15084693 | lifeskim:mentions | umls-concept:C2003903 | lld:lifeskim |
| pubmed-article:15084693 | lifeskim:mentions | umls-concept:C0220901 | lld:lifeskim |
| pubmed-article:15084693 | lifeskim:mentions | umls-concept:C1524063 | lld:lifeskim |
| pubmed-article:15084693 | pubmed:issue | 16 | lld:pubmed |
| pubmed-article:15084693 | pubmed:dateCreated | 2004-4-15 | lld:pubmed |
| pubmed-article:15084693 | pubmed:abstractText | In patients with acute myeloid leukemia (AML), the presence or absence of recurrent cytogenetic aberrations is used to identify the appropriate therapy. However, the current classification system does not fully reflect the molecular heterogeneity of the disease, and treatment stratification is difficult, especially for patients with intermediate-risk AML with a normal karyotype. | lld:pubmed |
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| pubmed-article:15084693 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15084693 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15084693 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:15084693 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15084693 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:15084693 | pubmed:issn | 1533-4406 | lld:pubmed |
| pubmed-article:15084693 | pubmed:author | pubmed-author:DöhnerHartmut... | lld:pubmed |
| pubmed-article:15084693 | pubmed:author | pubmed-author:PollackJonath... | lld:pubmed |
| pubmed-article:15084693 | pubmed:author | pubmed-author:FröhlingStefa... | lld:pubmed |
| pubmed-article:15084693 | pubmed:author | pubmed-author:SchlenkRichar... | lld:pubmed |
| pubmed-article:15084693 | pubmed:author | pubmed-author:DöhnerKonstan... | lld:pubmed |
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| pubmed-article:15084693 | pubmed:author | pubmed-author:BairEricE | lld:pubmed |
| pubmed-article:15084693 | pubmed:copyrightInfo | Copyright 2004 Massachusetts Medical Society | lld:pubmed |
| pubmed-article:15084693 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:15084693 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:15084693 | pubmed:volume | 350 | lld:pubmed |
| pubmed-article:15084693 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15084693 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15084693 | pubmed:pagination | 1605-16 | lld:pubmed |
| pubmed-article:15084693 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:15084693 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15084693 | pubmed:articleTitle | Use of gene-expression profiling to identify prognostic subclasses in adult acute myeloid leukemia. | lld:pubmed |
| pubmed-article:15084693 | pubmed:affiliation | Department of Pathology, Stanford University, Stanford, Calif, USA. | lld:pubmed |
| pubmed-article:15084693 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15084693 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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