| pubmed-article:1501130 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1501130 | lifeskim:mentions | umls-concept:C0010453 | lld:lifeskim |
| pubmed-article:1501130 | lifeskim:mentions | umls-concept:C0034721 | lld:lifeskim |
| pubmed-article:1501130 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:1501130 | lifeskim:mentions | umls-concept:C0027882 | lld:lifeskim |
| pubmed-article:1501130 | lifeskim:mentions | umls-concept:C0596901 | lld:lifeskim |
| pubmed-article:1501130 | lifeskim:mentions | umls-concept:C0019564 | lld:lifeskim |
| pubmed-article:1501130 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
| pubmed-article:1501130 | lifeskim:mentions | umls-concept:C0521116 | lld:lifeskim |
| pubmed-article:1501130 | pubmed:dateCreated | 1992-9-17 | lld:pubmed |
| pubmed-article:1501130 | pubmed:abstractText | 1. The currents underlying the graded impulses in small cultured hippocampal neurons from rat embryos were analysed under voltage-clamp conditions with the tight-seal whole-cell recording technique. 2. The leak and capacitative currents induced by a potential step were linearly related to the potential in the range studied (-60 to -100 mV). 3. With steps to potentials more positive than -40 mV, at least two different potential-activated currents were detected: an initial transient current and a delayed sustained one. In addition, 40% of the cells studied showed a delayed transient current. 4. The initial transient current showed sigmoid activation and roughly exponential inactivation. Its reversal potential depended on the Na+ concentration and was close to the Na+ equilibrium potential. Further, it was blocked by 3.0 microM-tetrodotoxin, and was abolished when choline was substituted for Na+ in the extracellular solution. We concluded that this current was carried mainly by Na+ ions. 5. The delayed sustained current showed sigmoid activation and almost no inactivation within 40 ms. The reversal potential was close to the K+ equilibrium potential. We concluded that this current was carried mainly by K+ ions. 6. The delayed transient current was outward in the potential range studied (-50 to +120 mV) and did not depend on the pipette Cl- concentration. It was assumed that this current was carried mainly by K+ ions. 7. A quantitative description of the initial transient and the delayed sustained currents was developed on the basis of earlier descriptions of excitable membranes. | lld:pubmed |
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| pubmed-article:1501130 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1501130 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1501130 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:1501130 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1501130 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:1501130 | pubmed:issn | 0022-3751 | lld:pubmed |
| pubmed-article:1501130 | pubmed:author | pubmed-author:ArhemPP | lld:pubmed |
| pubmed-article:1501130 | pubmed:author | pubmed-author:JohanssonSS | lld:pubmed |
| pubmed-article:1501130 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1501130 | pubmed:volume | 445 | lld:pubmed |
| pubmed-article:1501130 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1501130 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1501130 | pubmed:pagination | 141-56 | lld:pubmed |
| pubmed-article:1501130 | pubmed:dateRevised | 2010-9-7 | lld:pubmed |
| pubmed-article:1501130 | pubmed:meshHeading | pubmed-meshheading:1501130-... | lld:pubmed |
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| pubmed-article:1501130 | pubmed:meshHeading | pubmed-meshheading:1501130-... | lld:pubmed |
| pubmed-article:1501130 | pubmed:meshHeading | pubmed-meshheading:1501130-... | lld:pubmed |
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| pubmed-article:1501130 | pubmed:meshHeading | pubmed-meshheading:1501130-... | lld:pubmed |
| pubmed-article:1501130 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1501130 | pubmed:articleTitle | Membrane currents in small cultured rat hippocampal neurons: a voltage-clamp study. | lld:pubmed |
| pubmed-article:1501130 | pubmed:affiliation | Nobel Institute for Neurophysiology, Karolinska Institutet, Stockholm, Sweden. | lld:pubmed |
| pubmed-article:1501130 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1501130 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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