| pubmed-article:14657874 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:14657874 | lifeskim:mentions | umls-concept:C0087111 | lld:lifeskim |
| pubmed-article:14657874 | lifeskim:mentions | umls-concept:C0011306 | lld:lifeskim |
| pubmed-article:14657874 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:14657874 | lifeskim:mentions | umls-concept:C0966225 | lld:lifeskim |
| pubmed-article:14657874 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:14657874 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:14657874 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:14657874 | pubmed:dateCreated | 2003-12-5 | lld:pubmed |
| pubmed-article:14657874 | pubmed:abstractText | Dendritic cells (DCs) are potent antigen-presenting cells that express FcepsilonRI, the high-affinity IgE receptor. Although the downregulation of basophil FcepsilonRI during anti-IgE therapy with omalizumab is well documented, its effect on FcepsilonRI expression by DCs has not been reported. | lld:pubmed |
| pubmed-article:14657874 | pubmed:language | eng | lld:pubmed |
| pubmed-article:14657874 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14657874 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:14657874 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:14657874 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:14657874 | pubmed:issn | 0091-6749 | lld:pubmed |
| pubmed-article:14657874 | pubmed:author | pubmed-author:FosterBarbara... | lld:pubmed |
| pubmed-article:14657874 | pubmed:author | pubmed-author:PrussinCalman... | lld:pubmed |
| pubmed-article:14657874 | pubmed:author | pubmed-author:CasaleThomas... | lld:pubmed |
| pubmed-article:14657874 | pubmed:author | pubmed-author:LinHenryH | lld:pubmed |
| pubmed-article:14657874 | pubmed:author | pubmed-author:GriffithDanie... | lld:pubmed |
| pubmed-article:14657874 | pubmed:author | pubmed-author:BoeselKevin... | lld:pubmed |
| pubmed-article:14657874 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:14657874 | pubmed:volume | 112 | lld:pubmed |
| pubmed-article:14657874 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:14657874 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:14657874 | pubmed:pagination | 1147-54 | lld:pubmed |
| pubmed-article:14657874 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:14657874 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:14657874 | pubmed:articleTitle | Omalizumab treatment downregulates dendritic cell FcepsilonRI expression. | lld:pubmed |
| pubmed-article:14657874 | pubmed:affiliation | Laboratory of Allergic Diseases, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD, USA. | lld:pubmed |
| pubmed-article:14657874 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:14657874 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:14657874 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:14657874 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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