pubmed-article:14597946 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:14597946 | lifeskim:mentions | umls-concept:C0004388 | lld:lifeskim |
pubmed-article:14597946 | lifeskim:mentions | umls-concept:C0065374 | lld:lifeskim |
pubmed-article:14597946 | lifeskim:mentions | umls-concept:C0216784 | lld:lifeskim |
pubmed-article:14597946 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
pubmed-article:14597946 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:14597946 | lifeskim:mentions | umls-concept:C0264716 | lld:lifeskim |
pubmed-article:14597946 | lifeskim:mentions | umls-concept:C1707455 | lld:lifeskim |
pubmed-article:14597946 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:14597946 | pubmed:dateCreated | 2003-11-4 | lld:pubmed |
pubmed-article:14597946 | pubmed:abstractText | In chronic heart failure (CHF), the derangement of autonomic nervous system activity has a deep impact on the progression of the disease. It has been demonstrated that modulation of the renin-angiotensin aldosterone system (RAAS) increases autonomic control of heart rate and reduces adrenergic activity. We sought to evaluate, in CHF, the different effects of an ACE inhibitor (lisinopril) and of an AT1 receptor antagonist (valsartan) on heart rate variability, baroreflex sensitivity and norepinephrine plasma levels. | lld:pubmed |
pubmed-article:14597946 | pubmed:language | eng | lld:pubmed |
pubmed-article:14597946 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:14597946 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:14597946 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:14597946 | pubmed:month | Nov | lld:pubmed |
pubmed-article:14597946 | pubmed:issn | 1097-6744 | lld:pubmed |
pubmed-article:14597946 | pubmed:author | pubmed-author:MassariFrance... | lld:pubmed |
pubmed-article:14597946 | pubmed:author | pubmed-author:GuidaPietroP | lld:pubmed |
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pubmed-article:14597946 | pubmed:author | pubmed-author:FerrariRobert... | lld:pubmed |
pubmed-article:14597946 | pubmed:author | pubmed-author:CeconiClaudio... | lld:pubmed |
pubmed-article:14597946 | pubmed:author | pubmed-author:RomitoRoberta... | lld:pubmed |
pubmed-article:14597946 | pubmed:author | pubmed-author:De... | lld:pubmed |
pubmed-article:14597946 | pubmed:author | pubmed-author:FrancoliniGlo... | lld:pubmed |
pubmed-article:14597946 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:14597946 | pubmed:volume | 146 | lld:pubmed |
pubmed-article:14597946 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:14597946 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:14597946 | pubmed:pagination | E17 | lld:pubmed |
pubmed-article:14597946 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:14597946 | pubmed:year | 2003 | lld:pubmed |
pubmed-article:14597946 | pubmed:articleTitle | Comparison of the effect of valsartan and lisinopril on autonomic nervous system activity in chronic heart failure. | lld:pubmed |
pubmed-article:14597946 | pubmed:affiliation | Institute of Cardiology, University of Bari, Bari, Italy. | lld:pubmed |
pubmed-article:14597946 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:14597946 | pubmed:publicationType | Clinical Trial | lld:pubmed |
pubmed-article:14597946 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:14597946 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
pubmed-article:14597946 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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