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pubmed-article:14572785pubmed:abstractTextRecent evidence suggests that sepsis may induce an uncoupling of oxidative phosphorylation. The purpose of this study was to quantify temporal changes in hepatic oxygen consumption and cellular energy state with increasing severity of sepsis and thus assess the interrelationship of these parameters as either primary defect or compensatory response.lld:pubmed
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pubmed-article:14572785pubmed:articleTitleSepsis-induced failure of hepatic energy metabolism.lld:pubmed
pubmed-article:14572785pubmed:affiliationDepartment of Surgery, The University of Texas Medical Branch, Galveston, Texas, USA. dcgore@utmb.edulld:pubmed
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