| pubmed-article:14500528 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:14500528 | lifeskim:mentions | umls-concept:C0024198 | lld:lifeskim |
| pubmed-article:14500528 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:14500528 | lifeskim:mentions | umls-concept:C2936894 | lld:lifeskim |
| pubmed-article:14500528 | lifeskim:mentions | umls-concept:C0333348 | lld:lifeskim |
| pubmed-article:14500528 | lifeskim:mentions | umls-concept:C1327413 | lld:lifeskim |
| pubmed-article:14500528 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:14500528 | pubmed:dateCreated | 2003-9-22 | lld:pubmed |
| pubmed-article:14500528 | pubmed:abstractText | In a study of cytokine production ex vivo by Borrelia burgdorferi-stimulated peripheral blood mononuclear cells from 27 patients with culture-positive erythema migrans, production of inflammatory cytokines predominated, particularly gamma interferon and, to a lesser degree, tumor necrosis factor alpha. In contrast, with the exception of interleukin-13, anti-inflammatory cytokine production was negligible. Thus, B. burgdorferi antigens in early Lyme disease often induce a strong inflammatory response. | lld:pubmed |
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| pubmed-article:14500528 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:14500528 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:14500528 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:14500528 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:14500528 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:14500528 | pubmed:author | pubmed-author:SteereAllen... | lld:pubmed |
| pubmed-article:14500528 | pubmed:author | pubmed-author:DamleNitinN | lld:pubmed |
| pubmed-article:14500528 | pubmed:author | pubmed-author:MooreBrianB | lld:pubmed |
| pubmed-article:14500528 | pubmed:author | pubmed-author:SikandVijayV | lld:pubmed |
| pubmed-article:14500528 | pubmed:author | pubmed-author:GlicksteinLis... | lld:pubmed |
| pubmed-article:14500528 | pubmed:author | pubmed-author:BledsoeTaraT | lld:pubmed |
| pubmed-article:14500528 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:14500528 | pubmed:volume | 71 | lld:pubmed |
| pubmed-article:14500528 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:14500528 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:14500528 | pubmed:pagination | 6051-3 | lld:pubmed |
| pubmed-article:14500528 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:14500528 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:14500528 | pubmed:articleTitle | Inflammatory cytokine production predominates in early Lyme disease in patients with erythema migrans. | lld:pubmed |
| pubmed-article:14500528 | pubmed:affiliation | Division of Rheumatology/Immunology, New England Medical Center, Tufts University School of Medicine, Boston, MA 02111, USA. lglickstein@partners.org | lld:pubmed |
| pubmed-article:14500528 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:14500528 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:14500528 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:14500528 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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