| pubmed-article:1365532 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1365532 | lifeskim:mentions | umls-concept:C0031308 | lld:lifeskim |
| pubmed-article:1365532 | lifeskim:mentions | umls-concept:C0034793 | lld:lifeskim |
| pubmed-article:1365532 | lifeskim:mentions | umls-concept:C0300500 | lld:lifeskim |
| pubmed-article:1365532 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:1365532 | pubmed:dateCreated | 1995-2-9 | lld:pubmed |
| pubmed-article:1365532 | pubmed:abstractText | Phagocytosis is an essential component of host defense against invading pathogens. Although many cell types can ingest particulate material, "professional phagocytes" perform this function much more efficiently than other cells because they express unique membrane proteins and signal transduction mechanisms. This review addresses the current state of knowledge about the process of phagocytosis and its regulation. | lld:pubmed |
| pubmed-article:1365532 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1365532 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1365532 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1365532 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1365532 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:1365532 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1365532 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1365532 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1365532 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1365532 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:1365532 | pubmed:issn | 1056-2044 | lld:pubmed |
| pubmed-article:1365532 | pubmed:author | pubmed-author:BrownE JEJ | lld:pubmed |
| pubmed-article:1365532 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1365532 | pubmed:volume | 1 | lld:pubmed |
| pubmed-article:1365532 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1365532 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1365532 | pubmed:pagination | 63-70 | lld:pubmed |
| pubmed-article:1365532 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
| pubmed-article:1365532 | pubmed:meshHeading | pubmed-meshheading:1365532-... | lld:pubmed |
| pubmed-article:1365532 | pubmed:meshHeading | pubmed-meshheading:1365532-... | lld:pubmed |
| pubmed-article:1365532 | pubmed:meshHeading | pubmed-meshheading:1365532-... | lld:pubmed |
| pubmed-article:1365532 | pubmed:meshHeading | pubmed-meshheading:1365532-... | lld:pubmed |
| pubmed-article:1365532 | pubmed:meshHeading | pubmed-meshheading:1365532-... | lld:pubmed |
| pubmed-article:1365532 | pubmed:meshHeading | pubmed-meshheading:1365532-... | lld:pubmed |
| pubmed-article:1365532 | pubmed:meshHeading | pubmed-meshheading:1365532-... | lld:pubmed |
| pubmed-article:1365532 | pubmed:meshHeading | pubmed-meshheading:1365532-... | lld:pubmed |
| pubmed-article:1365532 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1365532 | pubmed:articleTitle | Complement receptors, adhesion, and phagocytosis. | lld:pubmed |
| pubmed-article:1365532 | pubmed:affiliation | Division of Infectious Diseases, Washington University School of Medicine, St. Louis, MO 63110. | lld:pubmed |
| pubmed-article:1365532 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1365532 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:1365532 | pubmed:publicationType | Review | lld:pubmed |
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