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| pubmed-article:1346576 | lifeskim:mentions | umls-concept:C0086860 | lld:lifeskim |
| pubmed-article:1346576 | lifeskim:mentions | umls-concept:C1880022 | lld:lifeskim |
| pubmed-article:1346576 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:1346576 | pubmed:dateCreated | 1992-3-16 | lld:pubmed |
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| pubmed-article:1346576 | pubmed:abstractText | The CD11b/CD18 heterodimeric surface antigen is expressed exclusively on human monocytes, macrophages, granulocytes, and natural killer cells. During differentiation of myeloid cell lines, CD11b steady state messenger RNA levels increase significantly; we show here that CD11b transcription rates increase commensurately. A 1.7-kb fragment of CD11b 5' flanking sequence directs expression of a reporter gene specifically in myeloid cell lines. Deletion analysis localizes elements directing high levels of tissue-specific reporter gene expression to the 412 bp proximal to the transcriptional start site. This sequence contains two consensus binding sites for Sp1, a GATA motif, and a purine-rich sequence that presents potential binding sites for members of the ets family of genes. Analysis of this promoter should result in the isolation of myeloid-specific transcription factors and the development of methods to direct the myeloid-specific expression of heterologous genes. | lld:pubmed |
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| pubmed-article:1346576 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1346576 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1346576 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:1346576 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1346576 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:1346576 | pubmed:issn | 0006-4971 | lld:pubmed |
| pubmed-article:1346576 | pubmed:author | pubmed-author:TenenD GDG | lld:pubmed |
| pubmed-article:1346576 | pubmed:author | pubmed-author:MuirPP | lld:pubmed |
| pubmed-article:1346576 | pubmed:author | pubmed-author:RosmarinA GAG | lld:pubmed |
| pubmed-article:1346576 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1346576 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:1346576 | pubmed:volume | 79 | lld:pubmed |
| pubmed-article:1346576 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1346576 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1346576 | pubmed:pagination | 865-70 | lld:pubmed |
| pubmed-article:1346576 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:1346576 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1346576 | pubmed:articleTitle | Characterization of the myeloid-specific CD11b promoter. | lld:pubmed |
| pubmed-article:1346576 | pubmed:affiliation | Hematology/Oncology Division, Beth Israel Hospital, Boston, MA 02215. | lld:pubmed |
| pubmed-article:1346576 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1346576 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:1346576 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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