pubmed-article:12974497 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12974497 | lifeskim:mentions | umls-concept:C0020097 | lld:lifeskim |
pubmed-article:12974497 | lifeskim:mentions | umls-concept:C0032667 | lld:lifeskim |
pubmed-article:12974497 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:12974497 | lifeskim:mentions | umls-concept:C0024934 | lld:lifeskim |
pubmed-article:12974497 | lifeskim:mentions | umls-concept:C0014499 | lld:lifeskim |
pubmed-article:12974497 | lifeskim:mentions | umls-concept:C0449445 | lld:lifeskim |
pubmed-article:12974497 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:12974497 | pubmed:dateCreated | 2003-9-16 | lld:pubmed |
pubmed-article:12974497 | pubmed:abstractText | Human T-cell Leukaemia Virus Type I (HTLV-I) is a retrovirus that causes Adult T-cell Leukaemia (ATL). The transmission routes of HTLV-I are (i) from infected mothers to their newborn babies, (ii) from infected males (husbands) to females (their wives) by long-term sexual intercourse, and (iii) from infected females (wives) to males (their husbands). Eshima et al. (2001) analysed a continuous-time HTLV-I model with no age structure in the population. In this paper, we consider the population dynamics of HTLV-I infection in a discrete-time mathematical model incorporating an age structure. The necessary and sufficient condition for the extinction of HTLV-I is derived from the mathematical model. A simulation of the HTLV-I infection based on the model demonstrates a rapid reduction of the HTLV-I infection proportion in Japan. | lld:pubmed |
pubmed-article:12974497 | pubmed:language | eng | lld:pubmed |
pubmed-article:12974497 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12974497 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:12974497 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12974497 | pubmed:month | Mar | lld:pubmed |
pubmed-article:12974497 | pubmed:issn | 1477-8599 | lld:pubmed |
pubmed-article:12974497 | pubmed:author | pubmed-author:EshimaNobuoki... | lld:pubmed |
pubmed-article:12974497 | pubmed:author | pubmed-author:TabataMinoruM | lld:pubmed |
pubmed-article:12974497 | pubmed:author | pubmed-author:OkadaTadashig... | lld:pubmed |
pubmed-article:12974497 | pubmed:author | pubmed-author:KarukayaShige... | lld:pubmed |
pubmed-article:12974497 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12974497 | pubmed:volume | 20 | lld:pubmed |
pubmed-article:12974497 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12974497 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12974497 | pubmed:pagination | 29-45 | lld:pubmed |
pubmed-article:12974497 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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pubmed-article:12974497 | pubmed:year | 2003 | lld:pubmed |
pubmed-article:12974497 | pubmed:articleTitle | Population dynamics of HTLV-I infection: a discrete-time mathematical epidemic model approach. | lld:pubmed |
pubmed-article:12974497 | pubmed:affiliation | Department of Biomathematics, Faculty of Medicine, Oita Medical University, Oita 879-5593, Japan. eshima@oita-med.ac.jp | lld:pubmed |
pubmed-article:12974497 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12974497 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |