| pubmed-article:1297314 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1297314 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
| pubmed-article:1297314 | lifeskim:mentions | umls-concept:C0225336 | lld:lifeskim |
| pubmed-article:1297314 | lifeskim:mentions | umls-concept:C0003483 | lld:lifeskim |
| pubmed-article:1297314 | lifeskim:mentions | umls-concept:C1258027 | lld:lifeskim |
| pubmed-article:1297314 | lifeskim:mentions | umls-concept:C1522492 | lld:lifeskim |
| pubmed-article:1297314 | pubmed:dateCreated | 1992-3-11 | lld:pubmed |
| pubmed-article:1297314 | pubmed:abstractText | Inhibition of tetrahydrobiopterin (H4biopterin) biosynthesis in endothelial cells almost completely abolished the agonist-induced formation of endothelium-derived relaxing factor (EDRF) (NO). This inhibitory effect could be antagonized when H4biopterin biosynthesis was restored by activating a salvage pathway. These data indicate that the formation of EDRF strictly depends on the presence of intracellular H4biopterin, which, in addition to Ca2+, may represent a further physiological and/or pathophysiological regulatory of endothelial NO synthases. | lld:pubmed |
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| pubmed-article:1297314 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:1297314 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1297314 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1297314 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:1297314 | pubmed:issn | 0264-6021 | lld:pubmed |
| pubmed-article:1297314 | pubmed:author | pubmed-author:SchmidtKK | lld:pubmed |
| pubmed-article:1297314 | pubmed:author | pubmed-author:KukovetzW RWR | lld:pubmed |
| pubmed-article:1297314 | pubmed:author | pubmed-author:MayerBB | lld:pubmed |
| pubmed-article:1297314 | pubmed:author | pubmed-author:WernerE RER | lld:pubmed |
| pubmed-article:1297314 | pubmed:author | pubmed-author:WachterHH | lld:pubmed |
| pubmed-article:1297314 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1297314 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:1297314 | pubmed:volume | 281 ( Pt 2) | lld:pubmed |
| pubmed-article:1297314 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1297314 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1297314 | pubmed:pagination | 297-300 | lld:pubmed |
| pubmed-article:1297314 | pubmed:dateRevised | 2010-9-7 | lld:pubmed |
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| pubmed-article:1297314 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1297314 | pubmed:articleTitle | Tetrahydrobiopterin-dependent formation of endothelium-derived relaxing factor (nitric oxide) in aortic endothelial cells. | lld:pubmed |
| pubmed-article:1297314 | pubmed:affiliation | Institut für Pharmakodynamik und Toxikologie, Universität Graz, Austria. | lld:pubmed |
| pubmed-article:1297314 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1297314 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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