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pubmed-article:12955405pubmed:abstractTextNitric oxide (NO) is known to act as a mediator of tissue injury as well as being a potent endogenous vasodilator. The functional and metabolic effects of NO on ischemia-reperfusion injury are still controversial. The aim of this study was to clarify the relationship between the degree of NO synthase (NOS) inhibition and the effects on ischemia-reperfusion injury.lld:pubmed
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pubmed-article:12955405pubmed:articleTitleThe dual effects of nitric oxide synthase inhibitors on ischemia-reperfusion injury in rat hearts.lld:pubmed
pubmed-article:12955405pubmed:affiliationSecond Dept. of Medicine, Kyoto Prefectural University of Medicine, Kawaramachi-Hirokoji, Kamigyo-ku, Kyoto 602-8566, Japan.lld:pubmed
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