| pubmed-article:12920062 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12920062 | lifeskim:mentions | umls-concept:C0007193 | lld:lifeskim |
| pubmed-article:12920062 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:12920062 | lifeskim:mentions | umls-concept:C0205245 | lld:lifeskim |
| pubmed-article:12920062 | lifeskim:mentions | umls-concept:C0031437 | lld:lifeskim |
| pubmed-article:12920062 | lifeskim:mentions | umls-concept:C1416877 | lld:lifeskim |
| pubmed-article:12920062 | lifeskim:mentions | umls-concept:C0686907 | lld:lifeskim |
| pubmed-article:12920062 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:12920062 | pubmed:dateCreated | 2003-8-15 | lld:pubmed |
| pubmed-article:12920062 | pubmed:abstractText | Mutations in the lamin A/C gene (LMNA) have been reported to be involved in dilated cardiomyopathy (DCM) associated with conduction system disease and/or skeletal myopathy. The aim of this study was to perform a mutational analysis of LMNA in a large white population of patients affected by dilated cardiomyopathy with or without associated symptoms. | lld:pubmed |
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| pubmed-article:12920062 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12920062 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12920062 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:12920062 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12920062 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12920062 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:12920062 | pubmed:issn | 1468-6244 | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:VillarsJJ | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:SchwartzKK | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:KomajdaMM | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:MillaireAA | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:BonneGG | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:BouchierCC | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:CharronPP | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:SébillonPP | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:Drouin-Garrau... | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:DesrumeauxGG | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:BidotL DLD | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:AhamedKK | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:BenaïcheAA | lld:pubmed |
| pubmed-article:12920062 | pubmed:author | pubmed-author:CharniotJ-CJC | lld:pubmed |
| pubmed-article:12920062 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:12920062 | pubmed:volume | 40 | lld:pubmed |
| pubmed-article:12920062 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12920062 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12920062 | pubmed:pagination | 560-7 | lld:pubmed |
| pubmed-article:12920062 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:12920062 | pubmed:meshHeading | pubmed-meshheading:12920062... | lld:pubmed |
| pubmed-article:12920062 | pubmed:meshHeading | pubmed-meshheading:12920062... | lld:pubmed |
| pubmed-article:12920062 | pubmed:meshHeading | pubmed-meshheading:12920062... | lld:pubmed |
| pubmed-article:12920062 | pubmed:meshHeading | pubmed-meshheading:12920062... | lld:pubmed |
| pubmed-article:12920062 | pubmed:meshHeading | pubmed-meshheading:12920062... | lld:pubmed |
| pubmed-article:12920062 | pubmed:meshHeading | pubmed-meshheading:12920062... | lld:pubmed |
| pubmed-article:12920062 | pubmed:meshHeading | pubmed-meshheading:12920062... | lld:pubmed |
| pubmed-article:12920062 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12920062 | pubmed:articleTitle | Expanding the phenotype of LMNA mutations in dilated cardiomyopathy and functional consequences of these mutations. | lld:pubmed |
| pubmed-article:12920062 | pubmed:affiliation | Laboratoire Génétique et Insuffisance Cardiaque, Association Claude Bernard/Université Paris VI, Groupe Hospitalier Pitié-Salpêtrière, Paris, France. sebilion@infobiogen.fr | lld:pubmed |
