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| pubmed-article:1284437 | lifeskim:mentions | umls-concept:C0025914 | lld:lifeskim |
| pubmed-article:1284437 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:1284437 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
| pubmed-article:1284437 | lifeskim:mentions | umls-concept:C0020429 | lld:lifeskim |
| pubmed-article:1284437 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:1284437 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:1284437 | pubmed:issue | 1-2 | lld:pubmed |
| pubmed-article:1284437 | pubmed:dateCreated | 1993-5-26 | lld:pubmed |
| pubmed-article:1284437 | pubmed:abstractText | Intrathecal injection of N-methyl-D-aspartate (NMDA) induces a short duration hyperalgesia in mice. An inhibitor of nitric oxide synthase (NOS), N omega-nitro-L-arginine methyl ester (L-NAME), administered either systemically or intrathecally, blocked the NMDA-induced hyperalgesia. This effect was partially reversed by the NOS substrate, L-arginine. Intrathecal hemoglobin mimicked the effects of L-NAME. Intrathecal injection of the NO-donating compounds, sodium nitroprusside (SNP) and hydroxylamine, resulted in a hyperalgesia that lasted 3 h and was reduced by coadministration of hemoglobin. Thus, nitric oxide production appears to mediate NMDA-induced hypersensitivity and may contribute to other forms of centrally induced hyperalgesia. | lld:pubmed |
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| pubmed-article:1284437 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:1284437 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1284437 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1284437 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:1284437 | pubmed:issn | 0304-3940 | lld:pubmed |
| pubmed-article:1284437 | pubmed:author | pubmed-author:HaleyJ EJE | lld:pubmed |
| pubmed-article:1284437 | pubmed:author | pubmed-author:WilcoxG LGL | lld:pubmed |
| pubmed-article:1284437 | pubmed:author | pubmed-author:KittoK FKF | lld:pubmed |
| pubmed-article:1284437 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1284437 | pubmed:day | 14 | lld:pubmed |
| pubmed-article:1284437 | pubmed:volume | 148 | lld:pubmed |
| pubmed-article:1284437 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1284437 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1284437 | pubmed:pagination | 1-5 | lld:pubmed |
| pubmed-article:1284437 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:1284437 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1284437 | pubmed:articleTitle | Involvement of nitric oxide in spinally mediated hyperalgesia in the mouse. | lld:pubmed |
| pubmed-article:1284437 | pubmed:affiliation | Department of Pharmacology, Graduate Program in Neuroscience, University of Minnesota, Minneapolis 55455. | lld:pubmed |
| pubmed-article:1284437 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1284437 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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