| pubmed-article:12806621 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12806621 | lifeskim:mentions | umls-concept:C0006826 | lld:lifeskim |
| pubmed-article:12806621 | lifeskim:mentions | umls-concept:C0085508 | lld:lifeskim |
| pubmed-article:12806621 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:12806621 | lifeskim:mentions | umls-concept:C0599840 | lld:lifeskim |
| pubmed-article:12806621 | lifeskim:mentions | umls-concept:C1167395 | lld:lifeskim |
| pubmed-article:12806621 | lifeskim:mentions | umls-concept:C0232567 | lld:lifeskim |
| pubmed-article:12806621 | lifeskim:mentions | umls-concept:C0591833 | lld:lifeskim |
| pubmed-article:12806621 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:12806621 | pubmed:dateCreated | 2003-6-13 | lld:pubmed |
| pubmed-article:12806621 | pubmed:abstractText | Helicobacter pylori cag(+) strains and high-expression host interleukin 1beta (IL-1beta) polymorphisms augment the risk for intestinal-type gastric adenocarcinoma, a malignancy that predominates in males. We examined the effects of an H. pylori cancer-associated determinant (cagE), IL-1beta, and host gender in a transgenic hypergastrinemic (INS-GAS) murine model of gastric carcinogenesis. | lld:pubmed |
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| pubmed-article:12806621 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12806621 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:12806621 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12806621 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:12806621 | pubmed:issn | 0016-5085 | lld:pubmed |
| pubmed-article:12806621 | pubmed:author | pubmed-author:PeekRichard... | lld:pubmed |
| pubmed-article:12806621 | pubmed:author | pubmed-author:KrishnaUmaU | lld:pubmed |
| pubmed-article:12806621 | pubmed:author | pubmed-author:WangTimothy... | lld:pubmed |
| pubmed-article:12806621 | pubmed:author | pubmed-author:FoxJames GJG | lld:pubmed |
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| pubmed-article:12806621 | pubmed:author | pubmed-author:TaylorNancyN | lld:pubmed |
| pubmed-article:12806621 | pubmed:author | pubmed-author:GeZhongmingZ | lld:pubmed |
| pubmed-article:12806621 | pubmed:author | pubmed-author:PoutahidisThe... | lld:pubmed |
| pubmed-article:12806621 | pubmed:author | pubmed-author:GausKristenK | lld:pubmed |
| pubmed-article:12806621 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12806621 | pubmed:volume | 124 | lld:pubmed |
| pubmed-article:12806621 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12806621 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12806621 | pubmed:pagination | 1879-90 | lld:pubmed |
| pubmed-article:12806621 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:12806621 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12806621 | pubmed:articleTitle | Host and microbial constituents influence Helicobacter pylori-induced cancer in a murine model of hypergastrinemia. | lld:pubmed |
| pubmed-article:12806621 | pubmed:affiliation | Division of Comparative Medicine, Massachusetts Institute of Technology, Cambridge, MA, USA. | lld:pubmed |
| pubmed-article:12806621 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12806621 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:12806621 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:12806621 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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