| pubmed-article:12699440 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12699440 | lifeskim:mentions | umls-concept:C0008059 | lld:lifeskim |
| pubmed-article:12699440 | lifeskim:mentions | umls-concept:C0205653 | lld:lifeskim |
| pubmed-article:12699440 | lifeskim:mentions | umls-concept:C0684224 | lld:lifeskim |
| pubmed-article:12699440 | lifeskim:mentions | umls-concept:C0001627 | lld:lifeskim |
| pubmed-article:12699440 | lifeskim:mentions | umls-concept:C0005823 | lld:lifeskim |
| pubmed-article:12699440 | lifeskim:mentions | umls-concept:C0852654 | lld:lifeskim |
| pubmed-article:12699440 | lifeskim:mentions | umls-concept:C0439611 | lld:lifeskim |
| pubmed-article:12699440 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:12699440 | pubmed:dateCreated | 2003-4-17 | lld:pubmed |
| pubmed-article:12699440 | pubmed:abstractText | Classical congenital adrenal hyperplasia (CAH) is characterized by a defect in cortisol and aldosterone secretion, adrenal hyperandrogenism, impaired adrenal medullary function and insulin insensitivity. The latter along with the increased tendency towards obesity raises questions whether other cardiovascular risk factors are altered in CAH. | lld:pubmed |
| pubmed-article:12699440 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12699440 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12699440 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12699440 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12699440 | pubmed:month | May | lld:pubmed |
| pubmed-article:12699440 | pubmed:issn | 0300-0664 | lld:pubmed |
| pubmed-article:12699440 | pubmed:author | pubmed-author:HindmarshPete... | lld:pubmed |
| pubmed-article:12699440 | pubmed:author | pubmed-author:CharmandariEv... | lld:pubmed |
| pubmed-article:12699440 | pubmed:author | pubmed-author:DattaniMehul... | lld:pubmed |
| pubmed-article:12699440 | pubmed:author | pubmed-author:RocheEdna FEF | lld:pubmed |
| pubmed-article:12699440 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12699440 | pubmed:volume | 58 | lld:pubmed |
| pubmed-article:12699440 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12699440 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12699440 | pubmed:pagination | 589-96 | lld:pubmed |
| pubmed-article:12699440 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:12699440 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12699440 | pubmed:articleTitle | Blood pressure in children and adolescents with congenital adrenal hyperplasia (21-hydroxylase deficiency): a preliminary report. | lld:pubmed |
| pubmed-article:12699440 | pubmed:affiliation | Centre for Human Growth and Maturation, London Centre for Paediatric Endocrinology, University College London, UK. | lld:pubmed |
| pubmed-article:12699440 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12699440 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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