pubmed-article:12569276 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12569276 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
pubmed-article:12569276 | lifeskim:mentions | umls-concept:C0015350 | lld:lifeskim |
pubmed-article:12569276 | lifeskim:mentions | umls-concept:C0003009 | lld:lifeskim |
pubmed-article:12569276 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
pubmed-article:12569276 | lifeskim:mentions | umls-concept:C0079284 | lld:lifeskim |
pubmed-article:12569276 | lifeskim:mentions | umls-concept:C1135196 | lld:lifeskim |
pubmed-article:12569276 | lifeskim:mentions | umls-concept:C0243076 | lld:lifeskim |
pubmed-article:12569276 | lifeskim:mentions | umls-concept:C0220905 | lld:lifeskim |
pubmed-article:12569276 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:12569276 | pubmed:dateCreated | 2003-2-5 | lld:pubmed |
pubmed-article:12569276 | pubmed:abstractText | Ventricular fibrosis plays a pivotal role in the development of diastolic heart failure and is a therapeutic target; however, the effects of pharmacological interventions on the extracellular matrix (ECM) regulatory system in diastolic heart failure remain to be clarified. | lld:pubmed |
pubmed-article:12569276 | pubmed:language | eng | lld:pubmed |
pubmed-article:12569276 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12569276 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:12569276 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12569276 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12569276 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12569276 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12569276 | pubmed:month | Feb | lld:pubmed |
pubmed-article:12569276 | pubmed:issn | 0263-6352 | lld:pubmed |
pubmed-article:12569276 | pubmed:author | pubmed-author:HoriMasatsugu... | lld:pubmed |
pubmed-article:12569276 | pubmed:author | pubmed-author:ManoToshiakiT | lld:pubmed |
pubmed-article:12569276 | pubmed:author | pubmed-author:YamamotoKazuh... | lld:pubmed |
pubmed-article:12569276 | pubmed:author | pubmed-author:MasuyamaTohru... | lld:pubmed |
pubmed-article:12569276 | pubmed:author | pubmed-author:SakataYasushi... | lld:pubmed |
pubmed-article:12569276 | pubmed:author | pubmed-author:NishikawaNaga... | lld:pubmed |
pubmed-article:12569276 | pubmed:author | pubmed-author:MiwaTakeshiT | lld:pubmed |
pubmed-article:12569276 | pubmed:author | pubmed-author:YoshidaJunich... | lld:pubmed |
pubmed-article:12569276 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12569276 | pubmed:volume | 21 | lld:pubmed |
pubmed-article:12569276 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12569276 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12569276 | pubmed:pagination | 437-44 | lld:pubmed |
pubmed-article:12569276 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:12569276 | pubmed:year | 2003 | lld:pubmed |
pubmed-article:12569276 | pubmed:articleTitle | Angiotensin II type 1 and endothelin type A receptor antagonists modulate the extracellular matrix regulatory system differently in diastolic heart failure. | lld:pubmed |
pubmed-article:12569276 | pubmed:affiliation | Department of Internal Medicine and Therapeutics, Osaka University Graduate School of Medicine and Genome Information Research Center, Osaka University, Suita, Japan. | lld:pubmed |
pubmed-article:12569276 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12569276 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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