| pubmed-article:12566371 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12566371 | lifeskim:mentions | umls-concept:C0022658 | lld:lifeskim |
| pubmed-article:12566371 | lifeskim:mentions | umls-concept:C0065374 | lld:lifeskim |
| pubmed-article:12566371 | lifeskim:mentions | umls-concept:C0178602 | lld:lifeskim |
| pubmed-article:12566371 | lifeskim:mentions | umls-concept:C0549634 | lld:lifeskim |
| pubmed-article:12566371 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:12566371 | lifeskim:mentions | umls-concept:C0205191 | lld:lifeskim |
| pubmed-article:12566371 | lifeskim:mentions | umls-concept:C1880371 | lld:lifeskim |
| pubmed-article:12566371 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:12566371 | pubmed:dateCreated | 2003-2-4 | lld:pubmed |
| pubmed-article:12566371 | pubmed:abstractText | Dyslipidemia frequently complicates chronic nephropathies and increases the risk of renal and cardiovascular events. This might be ameliorated by drugs, such as angiotensin-converting enzyme inhibitors, which effectively reduce proteinuria. | lld:pubmed |
| pubmed-article:12566371 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12566371 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12566371 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:12566371 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12566371 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12566371 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:12566371 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:12566371 | pubmed:author | pubmed-author:JazínE EEE | lld:pubmed |
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| pubmed-article:12566371 | pubmed:author | pubmed-author:MiseNaobumiN | lld:pubmed |
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| pubmed-article:12566371 | pubmed:author | pubmed-author:ArnoldiFederi... | lld:pubmed |
| pubmed-article:12566371 | pubmed:author | pubmed-author:PezzottaAnnaA | lld:pubmed |
| pubmed-article:12566371 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:12566371 | pubmed:day | 4 | lld:pubmed |
| pubmed-article:12566371 | pubmed:volume | 107 | lld:pubmed |
| pubmed-article:12566371 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12566371 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12566371 | pubmed:pagination | 586-92 | lld:pubmed |
| pubmed-article:12566371 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:12566371 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12566371 | pubmed:articleTitle | Diverse effects of increasing lisinopril doses on lipid abnormalities in chronic nephropathies. | lld:pubmed |
| pubmed-article:12566371 | pubmed:affiliation | Clinical Research Center for Rare Diseases Aldo and Cele Daccò, Mario Negri Institute, Bergamo, Italy. manuelap@marionegri.it | lld:pubmed |
| pubmed-article:12566371 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12566371 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:12566371 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:12566371 | pubmed:publicationType | Controlled Clinical Trial | lld:pubmed |
| pubmed-article:12566371 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
