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pubmed-article:12478193pubmed:abstractTextTriphasic changes in renal blood flow and ureteral pressure after unilateral ureteral obstruction have long been known. The contribution of nitric oxide to the decline in renal blood flow and ureteral pressure in unilateral ureteral obstruction was studied in this model using arginine infusion and by studying the effect of 2 inhibitors of nitric oxide synthase (NOS).lld:pubmed
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pubmed-article:12478193pubmed:articleTitleRenal hemodynamic and ureteral pressure changes in response to ureteral obstruction: the role of nitric oxide.lld:pubmed
pubmed-article:12478193pubmed:affiliationDepartment of Urology, James Buchanan Brady Foundation, Weill Medical College of Cornell University, New York, NY, USA.lld:pubmed
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