| pubmed-article:12426214 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C1956346 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C0225336 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C0003009 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C0068355 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C0529330 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C0332206 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C1512045 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:12426214 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:12426214 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:12426214 | pubmed:dateCreated | 2002-11-11 | lld:pubmed |
| pubmed-article:12426214 | pubmed:abstractText | Angiotensin II (Ang II)-mediated induction of vascular superoxide anion formation could contribute to the development of endothelial dysfunction, hypertension, and atherosclerosis. An NAD(P)H oxidase has been identified as a major endothelial source of superoxide anions. However, the molecular mechanism underlying the regulation of NAD(P)H oxidase activity in response to Ang II is not well understood. | lld:pubmed |
| pubmed-article:12426214 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12426214 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12426214 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12426214 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12426214 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:12426214 | pubmed:issn | 1524-4636 | lld:pubmed |
| pubmed-article:12426214 | pubmed:author | pubmed-author:QuinnMark TMT | lld:pubmed |
| pubmed-article:12426214 | pubmed:author | pubmed-author:HoltzJuergenJ | lld:pubmed |
| pubmed-article:12426214 | pubmed:author | pubmed-author:MorawietzHenn... | lld:pubmed |
| pubmed-article:12426214 | pubmed:author | pubmed-author:RueckschlossU... | lld:pubmed |
| pubmed-article:12426214 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:12426214 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:12426214 | pubmed:volume | 22 | lld:pubmed |
| pubmed-article:12426214 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12426214 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12426214 | pubmed:pagination | 1845-51 | lld:pubmed |
| pubmed-article:12426214 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:12426214 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12426214 | pubmed:articleTitle | Dose-dependent regulation of NAD(P)H oxidase expression by angiotensin II in human endothelial cells: protective effect of angiotensin II type 1 receptor blockade in patients with coronary artery disease. | lld:pubmed |
| pubmed-article:12426214 | pubmed:affiliation | Institute of Pathophysiology, Martin Luther University Halle-Wittenberg, Halle, Germany. | lld:pubmed |
| pubmed-article:12426214 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12426214 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:12426214 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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