pubmed-article:12234852 | rdf:type | pubmed:Citation | lld:pubmed |
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pubmed-article:12234852 | lifeskim:mentions | umls-concept:C0205147 | lld:lifeskim |
pubmed-article:12234852 | lifeskim:mentions | umls-concept:C0082329 | lld:lifeskim |
pubmed-article:12234852 | lifeskim:mentions | umls-concept:C0949782 | lld:lifeskim |
pubmed-article:12234852 | lifeskim:mentions | umls-concept:C1533693 | lld:lifeskim |
pubmed-article:12234852 | lifeskim:mentions | umls-concept:C1979958 | lld:lifeskim |
pubmed-article:12234852 | lifeskim:mentions | umls-concept:C0449943 | lld:lifeskim |
pubmed-article:12234852 | lifeskim:mentions | umls-concept:C0162802 | lld:lifeskim |
pubmed-article:12234852 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:12234852 | pubmed:dateCreated | 2002-9-17 | lld:pubmed |
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pubmed-article:12234852 | pubmed:abstractText | The mutations that are responsible for fluoroquinolone resistance in the gyrA, gyrB, parC, and parE genes of Salmonella enterica serovar Typhi and serovar Paratyphi A were investigated. The sequences of the quinolone resistance-determining region of the gyrA gene in clinical isolates which showed decreased susceptibilities to fluoroquinolones had a single mutation at either the Ser-83 or the Asp-87 codon, and no mutations were found in the gyrB, parC, and parE genes. | lld:pubmed |
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pubmed-article:12234852 | pubmed:language | eng | lld:pubmed |
pubmed-article:12234852 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12234852 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:12234852 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12234852 | pubmed:month | Oct | lld:pubmed |
pubmed-article:12234852 | pubmed:issn | 0066-4804 | lld:pubmed |
pubmed-article:12234852 | pubmed:author | pubmed-author:WatanabeHaruo... | lld:pubmed |
pubmed-article:12234852 | pubmed:author | pubmed-author:HiroseKenjiK | lld:pubmed |
pubmed-article:12234852 | pubmed:author | pubmed-author:EzakiTakayuki... | lld:pubmed |
pubmed-article:12234852 | pubmed:author | pubmed-author:KawamuraYoshi... | lld:pubmed |
pubmed-article:12234852 | pubmed:author | pubmed-author:TamuraKazumic... | lld:pubmed |
pubmed-article:12234852 | pubmed:author | pubmed-author:SagaraHirokoH | lld:pubmed |
pubmed-article:12234852 | pubmed:author | pubmed-author:HashimotoAiA | lld:pubmed |
pubmed-article:12234852 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12234852 | pubmed:volume | 46 | lld:pubmed |
pubmed-article:12234852 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12234852 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12234852 | pubmed:pagination | 3249-52 | lld:pubmed |
pubmed-article:12234852 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:12234852 | pubmed:meshHeading | pubmed-meshheading:12234852... | lld:pubmed |
pubmed-article:12234852 | pubmed:year | 2002 | lld:pubmed |
pubmed-article:12234852 | pubmed:articleTitle | DNA sequence analysis of DNA gyrase and DNA topoisomerase IV quinolone resistance-determining regions of Salmonella enterica serovar Typhi and serovar Paratyphi A. | lld:pubmed |
pubmed-article:12234852 | pubmed:affiliation | Department of Bacteriology, National Institute of Infectious Diseases, 1-23-1, Toyama, Shinjuku-ku, Tokyo, 162-8640, Japan. | lld:pubmed |
pubmed-article:12234852 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12234852 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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