| pubmed-article:12198693 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12198693 | lifeskim:mentions | umls-concept:C0301872 | lld:lifeskim |
| pubmed-article:12198693 | lifeskim:mentions | umls-concept:C1704410 | lld:lifeskim |
| pubmed-article:12198693 | lifeskim:mentions | umls-concept:C1707391 | lld:lifeskim |
| pubmed-article:12198693 | lifeskim:mentions | umls-concept:C0332120 | lld:lifeskim |
| pubmed-article:12198693 | lifeskim:mentions | umls-concept:C1517945 | lld:lifeskim |
| pubmed-article:12198693 | lifeskim:mentions | umls-concept:C1291775 | lld:lifeskim |
| pubmed-article:12198693 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:12198693 | pubmed:dateCreated | 2002-8-28 | lld:pubmed |
| pubmed-article:12198693 | pubmed:abstractText | Previous studies in Crohn's disease suggest global loss of tolerance with sonicated bacteria preparations containing hundreds of antigens. Monoassociation studies show that a solitary bacterium can induce colitis in one animal model, whereas another is responsible in other models. Among patients with Crohn's disease, serum responses have been documented to microbial and autoantigens (antibodies to the Escherichia coli outer-membrane porin C and the Pseudomonas fluorescens-associated sequence I2, antisaccharomyces cerevisiae antibody (ASCA), and perinuclear antineutrophil cytoplasmic antibodies). Our aim was to determine whether there are heterogeneous responses to these specific antigens. | lld:pubmed |
| pubmed-article:12198693 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12198693 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12198693 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:12198693 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12198693 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:12198693 | pubmed:issn | 0016-5085 | lld:pubmed |
| pubmed-article:12198693 | pubmed:author | pubmed-author:BraunJonathan... | lld:pubmed |
| pubmed-article:12198693 | pubmed:author | pubmed-author:YangHuiyingH | lld:pubmed |
| pubmed-article:12198693 | pubmed:author | pubmed-author:TarganStephan... | lld:pubmed |
| pubmed-article:12198693 | pubmed:author | pubmed-author:LinYing-ChaoY... | lld:pubmed |
| pubmed-article:12198693 | pubmed:author | pubmed-author:LandersCarol... | lld:pubmed |
| pubmed-article:12198693 | pubmed:author | pubmed-author:CohavyOfferO | lld:pubmed |
| pubmed-article:12198693 | pubmed:author | pubmed-author:MisraRajeevR | lld:pubmed |
| pubmed-article:12198693 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12198693 | pubmed:volume | 123 | lld:pubmed |
| pubmed-article:12198693 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12198693 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12198693 | pubmed:pagination | 689-99 | lld:pubmed |
| pubmed-article:12198693 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:12198693 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12198693 | pubmed:articleTitle | Selected loss of tolerance evidenced by Crohn's disease-associated immune responses to auto- and microbial antigens. | lld:pubmed |
| pubmed-article:12198693 | pubmed:affiliation | Cedars-Sinai Medical Center Inflammatory Bowel Disease Center, Los Angeles, California, USA. | lld:pubmed |
| pubmed-article:12198693 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12198693 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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