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pubmed-article:12135940 | lifeskim:mentions | umls-concept:C1416562 | lld:lifeskim |
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pubmed-article:12135940 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
pubmed-article:12135940 | lifeskim:mentions | umls-concept:C1705248 | lld:lifeskim |
pubmed-article:12135940 | lifeskim:mentions | umls-concept:C0521116 | lld:lifeskim |
pubmed-article:12135940 | lifeskim:mentions | umls-concept:C0302891 | lld:lifeskim |
pubmed-article:12135940 | lifeskim:mentions | umls-concept:C1548799 | lld:lifeskim |
pubmed-article:12135940 | lifeskim:mentions | umls-concept:C1524073 | lld:lifeskim |
pubmed-article:12135940 | lifeskim:mentions | umls-concept:C0449432 | lld:lifeskim |
pubmed-article:12135940 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:12135940 | pubmed:dateCreated | 2002-7-23 | lld:pubmed |
pubmed-article:12135940 | pubmed:abstractText | The transient outward potassium current (I(to)) encoded by the Kv4 family of potassium channels is important in the repolarization of cardiac myocytes. KChIPs are a recently identified group of Ca2+-binding accessory subunits that modulate Kv4-encoded currents. KChIP2 is the only family member expressed in the heart. | lld:pubmed |
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pubmed-article:12135940 | pubmed:language | eng | lld:pubmed |
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pubmed-article:12135940 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:12135940 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12135940 | pubmed:month | Jul | lld:pubmed |
pubmed-article:12135940 | pubmed:issn | 1524-4539 | lld:pubmed |
pubmed-article:12135940 | pubmed:author | pubmed-author:TomaselliGord... | lld:pubmed |
pubmed-article:12135940 | pubmed:author | pubmed-author:WuRichard CRC | lld:pubmed |
pubmed-article:12135940 | pubmed:author | pubmed-author:AnW FrankWF | lld:pubmed |
pubmed-article:12135940 | pubmed:author | pubmed-author:DeschênesIsab... | lld:pubmed |
pubmed-article:12135940 | pubmed:author | pubmed-author:DiSilvestreDe... | lld:pubmed |
pubmed-article:12135940 | pubmed:author | pubmed-author:JuangGeorge... | lld:pubmed |
pubmed-article:12135940 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:12135940 | pubmed:day | 23 | lld:pubmed |
pubmed-article:12135940 | pubmed:volume | 106 | lld:pubmed |
pubmed-article:12135940 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12135940 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12135940 | pubmed:pagination | 423-9 | lld:pubmed |
pubmed-article:12135940 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:12135940 | pubmed:year | 2002 | lld:pubmed |
pubmed-article:12135940 | pubmed:articleTitle | Regulation of Kv4.3 current by KChIP2 splice variants: a component of native cardiac I(to)? | lld:pubmed |
pubmed-article:12135940 | pubmed:affiliation | Department of Medicine, Institute of Molecular Cardiobiology, Division of Cardiology, Johns Hopkins University, Baltimore, Md 21205, USA. | lld:pubmed |
pubmed-article:12135940 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12135940 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:12135940 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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