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| pubmed-article:12135675 | lifeskim:mentions | umls-concept:C0023467 | lld:lifeskim |
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| pubmed-article:12135675 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:12135675 | lifeskim:mentions | umls-concept:C0018270 | lld:lifeskim |
| pubmed-article:12135675 | lifeskim:mentions | umls-concept:C1268567 | lld:lifeskim |
| pubmed-article:12135675 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:12135675 | lifeskim:mentions | umls-concept:C0237477 | lld:lifeskim |
| pubmed-article:12135675 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:12135675 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:12135675 | pubmed:dateCreated | 2002-7-23 | lld:pubmed |
| pubmed-article:12135675 | pubmed:abstractText | Angiogenesis, the process of new blood vessel formation, is a critical process during growth and metastasis of solid tumors and might also represent a promising therapeutical target in patients with acute myeloid leukemia (AML). | lld:pubmed |
| pubmed-article:12135675 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12135675 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12135675 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:12135675 | pubmed:issn | 0301-472X | lld:pubmed |
| pubmed-article:12135675 | pubmed:author | pubmed-author:HiddemannWolf... | lld:pubmed |
| pubmed-article:12135675 | pubmed:author | pubmed-author:SpiekermannKa... | lld:pubmed |
| pubmed-article:12135675 | pubmed:author | pubmed-author:FaberFlorianF | lld:pubmed |
| pubmed-article:12135675 | pubmed:author | pubmed-author:VoswinckelRob... | lld:pubmed |
| pubmed-article:12135675 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12135675 | pubmed:volume | 30 | lld:pubmed |
| pubmed-article:12135675 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12135675 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12135675 | pubmed:pagination | 767-73 | lld:pubmed |
| pubmed-article:12135675 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:12135675 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12135675 | pubmed:articleTitle | The protein tyrosine kinase inhibitor SU5614 inhibits VEGF-induced endothelial cell sprouting and induces growth arrest and apoptosis by inhibition of c-kit in AML cells. | lld:pubmed |
| pubmed-article:12135675 | pubmed:affiliation | Department of Medicine III, University Hospital Grosshadern, Clinical Cooperative Group Leukemia, GSF National Research Center for Environment and Health, Munich, Germany. spiekermann@gsf.de | lld:pubmed |
| pubmed-article:12135675 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12135675 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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