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pubmed-article:12106779pubmed:abstractTextThe regulation of noradrenaline (NA) release by endogenous endothelium-derived compounds was investigated in the isolated guinea pig pulmonary artery preloaded with [3H]NA. The radioactivity uptake, the basal and electrical field stimulation (10 Hz, 2 ms, 360 shocks) evoked release of [(3)H]NA was similar in arteries with intact endothelium and after removal of the endothelium. The wide selectivity nitric oxide (NO) synthase inhibitor N-omega-nitro-L-arginine (100 microM) did not affect significantly the basal and stimulation-evoked release of [(3)H]NA in control and endothelium-denuded preparations. These results indicate that neither endogenous NO nor other compounds derived from the endothelium have substantial influence on the NA outflow from sympathetic nerves innervating the pulmonary artery.lld:pubmed
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pubmed-article:12106779pubmed:pagination279-83lld:pubmed
pubmed-article:12106779pubmed:dateRevised2006-11-15lld:pubmed
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pubmed-article:12106779pubmed:articleTitleLack of prejunctional modulation of noradrenaline release by endogenous nitric oxide in guinea pig pulmonary artery.lld:pubmed
pubmed-article:12106779pubmed:affiliationUniversity of Medicine and Pharmacy, Tirgu-Mures, Romania.lld:pubmed
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