| pubmed-article:12049617 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12049617 | lifeskim:mentions | umls-concept:C0025329 | lld:lifeskim |
| pubmed-article:12049617 | lifeskim:mentions | umls-concept:C0206162 | lld:lifeskim |
| pubmed-article:12049617 | lifeskim:mentions | umls-concept:C0312418 | lld:lifeskim |
| pubmed-article:12049617 | lifeskim:mentions | umls-concept:C0036667 | lld:lifeskim |
| pubmed-article:12049617 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:12049617 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:12049617 | pubmed:dateCreated | 2002-6-6 | lld:pubmed |
| pubmed-article:12049617 | pubmed:abstractText | The menstrual cycle provokes several physiological changes that could influence autonomic regulatory mechanisms. We studied the carotid-cardiac baroreflex in ten healthy young women on four occasions over the course of their menstrual cycles (days 0-8, 9-14, 15-20 and 21-25). We drew blood during each session for analysis of oestrogen, progesterone and noradrenaline (norepinephrine) levels, and assessed carotid-cardiac baroreflex function by analysing R-R interval responses to graded neck pressure sequences. Oestrogen levels followed a classical two-peak (cubic) response, with elevated levels on days 9-14 and 21-25 compared with days 0-8 and 15-20 (P=0.0032), while progesterone levels increased exponentially from days 9-14 to days 21-25 (P=0.0063). Noradrenaline levels increased from an average of 137 pg/ml during the first three measurement periods to 199 pg/ml during days 21-25 (P=0.0456). Carotid-cardiac baroreflex gain and operational point were not statistically different at any of the time points during the menstrual cycle (P> or =0.18). These findings are consistent with the notion that beat-to-beat vagal-cardiac regulation does not change over the course of the normal menstrual cycle. | lld:pubmed |
| pubmed-article:12049617 | pubmed:keyword | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12049617 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12049617 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12049617 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:12049617 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12049617 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12049617 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12049617 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12049617 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:12049617 | pubmed:issn | 0143-5221 | lld:pubmed |
| pubmed-article:12049617 | pubmed:author | pubmed-author:CookeWilliam... | lld:pubmed |
| pubmed-article:12049617 | pubmed:author | pubmed-author:EckbergDwain... | lld:pubmed |
| pubmed-article:12049617 | pubmed:author | pubmed-author:LudwigDavid... | lld:pubmed |
| pubmed-article:12049617 | pubmed:author | pubmed-author:HoggPaul SPS | lld:pubmed |
| pubmed-article:12049617 | pubmed:author | pubmed-author:ConvertinoVic... | lld:pubmed |
| pubmed-article:12049617 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12049617 | pubmed:volume | 102 | lld:pubmed |
| pubmed-article:12049617 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12049617 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12049617 | pubmed:pagination | 639-44 | lld:pubmed |
| pubmed-article:12049617 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:meshHeading | pubmed-meshheading:12049617... | lld:pubmed |
| pubmed-article:12049617 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12049617 | pubmed:articleTitle | Does the menstrual cycle influence the sensitivity of vagally mediated baroreflexes? | lld:pubmed |
| pubmed-article:12049617 | pubmed:affiliation | Department of Biomedical Engineering, Michigan Technological University, Houghton 49931, USA. whcooke@mtu.edu | lld:pubmed |
| pubmed-article:12049617 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12049617 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:12049617 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
