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| pubmed-article:12031912 | lifeskim:mentions | umls-concept:C1533585 | lld:lifeskim |
| pubmed-article:12031912 | lifeskim:mentions | umls-concept:C0086661 | lld:lifeskim |
| pubmed-article:12031912 | lifeskim:mentions | umls-concept:C0023467 | lld:lifeskim |
| pubmed-article:12031912 | lifeskim:mentions | umls-concept:C0040715 | lld:lifeskim |
| pubmed-article:12031912 | lifeskim:mentions | umls-concept:C1514559 | lld:lifeskim |
| pubmed-article:12031912 | lifeskim:mentions | umls-concept:C0332197 | lld:lifeskim |
| pubmed-article:12031912 | lifeskim:mentions | umls-concept:C1333214 | lld:lifeskim |
| pubmed-article:12031912 | lifeskim:mentions | umls-concept:C1449575 | lld:lifeskim |
| pubmed-article:12031912 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:12031912 | pubmed:dateCreated | 2002-5-28 | lld:pubmed |
| pubmed-article:12031912 | pubmed:abstractText | Translocation-associated gene fusions are well recognized in acute myeloid leukemia. Other molecular genetic changes are less well known. The novel cDNA technology has opened the avenue to large-scale gene expression analysis. Our aim was to perform cDNA microarray analysis of acute myeloid leukemia (AML). | lld:pubmed |
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| pubmed-article:12031912 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12031912 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12031912 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:12031912 | pubmed:issn | 0390-6078 | lld:pubmed |
| pubmed-article:12031912 | pubmed:author | pubmed-author:VihinenMaunoM | lld:pubmed |
| pubmed-article:12031912 | pubmed:author | pubmed-author:KnuutilaSakar... | lld:pubmed |
| pubmed-article:12031912 | pubmed:author | pubmed-author:LarramendyMar... | lld:pubmed |
| pubmed-article:12031912 | pubmed:author | pubmed-author:OllilaJuhaJ | lld:pubmed |
| pubmed-article:12031912 | pubmed:author | pubmed-author:NagyBálintB | lld:pubmed |
| pubmed-article:12031912 | pubmed:author | pubmed-author:NiiniTarjaT | lld:pubmed |
| pubmed-article:12031912 | pubmed:author | pubmed-author:ElonenErkkiE | lld:pubmed |
| pubmed-article:12031912 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12031912 | pubmed:volume | 87 | lld:pubmed |
| pubmed-article:12031912 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12031912 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12031912 | pubmed:pagination | 569-77 | lld:pubmed |
| pubmed-article:12031912 | pubmed:dateRevised | 2010-5-26 | lld:pubmed |
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| pubmed-article:12031912 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12031912 | pubmed:articleTitle | Overexpression of translocation-associated fusion genes of FGFRI, MYC, NPMI, and DEK, but absence of the translocations in acute myeloid leukemia. A microarray analysis. | lld:pubmed |
| pubmed-article:12031912 | pubmed:affiliation | Department of Pathology, Haartman Institute, Helsinki University Central Hospital, FIN-00014 University of Helsinki, Haartmaninkatu 3, Helsinki, Finland. | lld:pubmed |
| pubmed-article:12031912 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12031912 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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