pubmed-article:12015366 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12015366 | lifeskim:mentions | umls-concept:C0020538 | lld:lifeskim |
pubmed-article:12015366 | lifeskim:mentions | umls-concept:C0681876 | lld:lifeskim |
pubmed-article:12015366 | lifeskim:mentions | umls-concept:C0026606 | lld:lifeskim |
pubmed-article:12015366 | lifeskim:mentions | umls-concept:C0005823 | lld:lifeskim |
pubmed-article:12015366 | lifeskim:mentions | umls-concept:C1413931 | lld:lifeskim |
pubmed-article:12015366 | lifeskim:mentions | umls-concept:C0016132 | lld:lifeskim |
pubmed-article:12015366 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:12015366 | pubmed:dateCreated | 2002-5-16 | lld:pubmed |
pubmed-article:12015366 | pubmed:abstractText | The study evaluated the association of the insertion/deletion polymorphism of the angiotensin-converting enzyme gene (ACE I/D) with self-reported moderate-intensity leisure time physical activity (MILTPA), arterial blood pressure (BP) and history of hypertension (HT). A representative population-based sample of 721 middle-aged adults (358 women) from two areas of Finland was genotyped for the ACE I/D. After exclusion criteria were applied, 455 subjects (288 women) were selected for the analysis. The distribution of the ACE I/D genotypes did not differ significantly among frequent vs. nonfrequent MILTPA groups (chi(2) = 2.556; df = 2; P value = 0.279). The main predictors of BP were male gender, age, body mass index, and arterial pulse. Additionally, tobacco smoking and alcohol consumption also had a significant main effect on diastolic BP. HT was significantly more frequent in subjects with obesity, family history of cardiovascular disease, or lower educational level. As for BP, neither ACE I/D nor MILTPA was associated with HT. The study confirmed recent reports from population-based studies of no association between ACE I/D and physical fitness. The study also confirmed a lack of association between ACE I/D and BP or HT. | lld:pubmed |
pubmed-article:12015366 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12015366 | pubmed:language | eng | lld:pubmed |
pubmed-article:12015366 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12015366 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:12015366 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12015366 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12015366 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12015366 | pubmed:month | Jun | lld:pubmed |
pubmed-article:12015366 | pubmed:issn | 8750-7587 | lld:pubmed |
pubmed-article:12015366 | pubmed:author | pubmed-author:TuomilehtoJaa... | lld:pubmed |
pubmed-article:12015366 | pubmed:author | pubmed-author:NissinenAulik... | lld:pubmed |
pubmed-article:12015366 | pubmed:author | pubmed-author:FuentesRicard... | lld:pubmed |
pubmed-article:12015366 | pubmed:author | pubmed-author:PerolaMarkusM | lld:pubmed |
pubmed-article:12015366 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12015366 | pubmed:volume | 92 | lld:pubmed |
pubmed-article:12015366 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12015366 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12015366 | pubmed:pagination | 2508-12 | lld:pubmed |
pubmed-article:12015366 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:meshHeading | pubmed-meshheading:12015366... | lld:pubmed |
pubmed-article:12015366 | pubmed:year | 2002 | lld:pubmed |
pubmed-article:12015366 | pubmed:articleTitle | ACE gene and physical activity, blood pressure, and hypertension: a population study in Finland. | lld:pubmed |
pubmed-article:12015366 | pubmed:affiliation | Department of Public Health and General Practice, Faculty of Medicine, University of Kuopio, FIN-70211 Kuopio, Finland. ricardo.fuentes@messi.uku.fi | lld:pubmed |
pubmed-article:12015366 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12015366 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
entrez-gene:1636 | entrezgene:pubmed | pubmed-article:12015366 | lld:entrezgene |
http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:12015366 | lld:entrezgene |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:12015366 | lld:pubmed |