| pubmed-article:12009565 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12009565 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:12009565 | lifeskim:mentions | umls-concept:C1155266 | lld:lifeskim |
| pubmed-article:12009565 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:12009565 | lifeskim:mentions | umls-concept:C1363844 | lld:lifeskim |
| pubmed-article:12009565 | pubmed:issue | 12-13 | lld:pubmed |
| pubmed-article:12009565 | pubmed:dateCreated | 2002-5-15 | lld:pubmed |
| pubmed-article:12009565 | pubmed:abstractText | Cytokines produced by innate immune response can profoundly influence the subsequent adaptive immunity. IL-15 and -18 are two of several mediators produced by macrophages that perform such a function. Here we present recent data mainly from our own laboratory illustrating the important role of IL-15 and -18 in the induction and perpetuation of chronic inflammation during experimental and clinical rheumatoid synovitis. | lld:pubmed |
| pubmed-article:12009565 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12009565 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12009565 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:12009565 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12009565 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12009565 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12009565 | pubmed:month | May | lld:pubmed |
| pubmed-article:12009565 | pubmed:issn | 0161-5890 | lld:pubmed |
| pubmed-article:12009565 | pubmed:author | pubmed-author:LiewFoo YFY | lld:pubmed |
| pubmed-article:12009565 | pubmed:author | pubmed-author:McInnesIain... | lld:pubmed |
| pubmed-article:12009565 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12009565 | pubmed:volume | 38 | lld:pubmed |
| pubmed-article:12009565 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12009565 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12009565 | pubmed:pagination | 887-90 | lld:pubmed |
| pubmed-article:12009565 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:meshHeading | pubmed-meshheading:12009565... | lld:pubmed |
| pubmed-article:12009565 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12009565 | pubmed:articleTitle | The role of innate mediators in inflammatory response. | lld:pubmed |
| pubmed-article:12009565 | pubmed:affiliation | Department of Immunology and Bacteriology, University of Glasgow, G11 6NT, UK. fyllh@clinmed.gla.ac.uk | lld:pubmed |
| pubmed-article:12009565 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12009565 | pubmed:publicationType | Review | lld:pubmed |
| pubmed-article:12009565 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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