| pubmed-article:11993309 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11993309 | lifeskim:mentions | umls-concept:C0008972 | lld:lifeskim |
| pubmed-article:11993309 | lifeskim:mentions | umls-concept:C0004331 | lld:lifeskim |
| pubmed-article:11993309 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:11993309 | lifeskim:mentions | umls-concept:C1710265 | lld:lifeskim |
| pubmed-article:11993309 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:11993309 | pubmed:dateCreated | 2002-5-7 | lld:pubmed |
| pubmed-article:11993309 | pubmed:abstractText | The electrocardiographic patterns recorded from seven patients with isorhythmic A-V dissociation fall into two distinct groups. In pattern I, the P wave fluctuates cyclically back and forth across the QRS complex. The mechanism responsible for this type of A-V synchronization represents a typical biologic feedback control system. The P-R interval is a determinant of stroke volume, which in turn influences the arterial blood pressure. The blood pressure has an inverse effect on the discharge frequency of the S-A node through the baroreceptor reflex. The S-A nodal frequency then affects the P-R interval, to close the feedback loop. In pattern II, the P wave is in a fairly constant position relative to the QRS complex. It is usually coincident with the QRS complex or appears on the ST segment or first half of the T wave. The mechanism producing synchronization in pattern II type of isorhythmic dissociation has not been established conclusively. | lld:pubmed |
| pubmed-article:11993309 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11993309 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11993309 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11993309 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:11993309 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11993309 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:11993309 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:11993309 | pubmed:author | pubmed-author:LevyM NMN | lld:pubmed |
| pubmed-article:11993309 | pubmed:author | pubmed-author:EdflsteinJJ | lld:pubmed |
| pubmed-article:11993309 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:11993309 | pubmed:volume | 42 | lld:pubmed |
| pubmed-article:11993309 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11993309 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11993309 | pubmed:pagination | 689-99 | lld:pubmed |
| pubmed-article:11993309 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
| pubmed-article:11993309 | pubmed:meshHeading | pubmed-meshheading:11993309... | lld:pubmed |
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| pubmed-article:11993309 | pubmed:year | 1970 | lld:pubmed |
| pubmed-article:11993309 | pubmed:articleTitle | The mechanism of synchronization in isorhythmic A-V dissociation. II. Clinical studies. | lld:pubmed |
| pubmed-article:11993309 | pubmed:affiliation | Departments of Investigative Medicine and Cardiology, Mt. Sinai Hospital, Cleveland, Ohio, USA. | lld:pubmed |
| pubmed-article:11993309 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11993309 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:11993309 | lld:pubmed |
