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pubmed-article:11966527pubmed:abstractTextEndogenous prostaglandins (PGs) are considered to play a pivotal role in maintaining the mucosal integrity of the stomach after injury. In the present study, we evaluated the mucosal ulcerogenic and mucosal blood flow (GMBF) responses in the stomach after damage by taurocholate (TC) in knockout mice lacking EP1 or EP3 receptors.lld:pubmed
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pubmed-article:11966527pubmed:dateRevised2010-11-18lld:pubmed
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pubmed-article:11966527pubmed:articleTitleGastric ulcerogenic responses following barrier disruption in knockout mice lacking prostaglandin EP1 receptors.lld:pubmed
pubmed-article:11966527pubmed:affiliationDepartment of Pharmacology and Experimental Therapeutics, Kyoto Pharmaceutical University, Misasagi, Yamashina, Japan. takeuchi@mb.kyoto-phu.ac.jplld:pubmed
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