| pubmed-article:11931222 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11931222 | lifeskim:mentions | umls-concept:C1522384 | lld:lifeskim |
| pubmed-article:11931222 | lifeskim:mentions | umls-concept:C0008633 | lld:lifeskim |
| pubmed-article:11931222 | lifeskim:mentions | umls-concept:C0205171 | lld:lifeskim |
| pubmed-article:11931222 | pubmed:dateCreated | 2002-4-4 | lld:pubmed |
| pubmed-article:11931222 | pubmed:abstractText | Just as homology can trigger a chain of events as described in many of the chapters of this volume, sometimes a lack of homology causes a crisis of a different sort. So it is for the single X chromosome in XY males in many species. Divergent sex chromosome pairs, such as the X and Y chromosomes in mammals and in fruit flies, are thought to have evolved from homologous autosomes. During evolution, the Y chromosome has retained little coding capacity, leaving the male with reduced gene dosage for many functions encoded by the X chromosome. In this chapter we focus on dosage compensation in Drosophila, in which most X-linked genes are upregulated by a male-specific ribonucleoprotein complex. This complex is thought to recognize the X chromosome through approximately 35 dispersed chromatin entry sites and then spread in cis to dosage compensate most genes on the X chromosome. | lld:pubmed |
| pubmed-article:11931222 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11931222 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11931222 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11931222 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11931222 | pubmed:issn | 0065-2660 | lld:pubmed |
| pubmed-article:11931222 | pubmed:author | pubmed-author:MellerVictori... | lld:pubmed |
| pubmed-article:11931222 | pubmed:author | pubmed-author:KurodaMitzi... | lld:pubmed |
| pubmed-article:11931222 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11931222 | pubmed:volume | 46 | lld:pubmed |
| pubmed-article:11931222 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11931222 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11931222 | pubmed:pagination | 1-24 | lld:pubmed |
| pubmed-article:11931222 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:11931222 | pubmed:meshHeading | pubmed-meshheading:11931222... | lld:pubmed |
| pubmed-article:11931222 | pubmed:meshHeading | pubmed-meshheading:11931222... | lld:pubmed |
| pubmed-article:11931222 | pubmed:meshHeading | pubmed-meshheading:11931222... | lld:pubmed |
| pubmed-article:11931222 | pubmed:meshHeading | pubmed-meshheading:11931222... | lld:pubmed |
| pubmed-article:11931222 | pubmed:meshHeading | pubmed-meshheading:11931222... | lld:pubmed |
| pubmed-article:11931222 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:11931222 | pubmed:articleTitle | Sex and the single chromosome. | lld:pubmed |
| pubmed-article:11931222 | pubmed:affiliation | Department of Biology, Tufts University, Medford, Massachusetts 02155, USA. | lld:pubmed |
| pubmed-article:11931222 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11931222 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:11931222 | pubmed:publicationType | Review | lld:pubmed |
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