| pubmed-article:11920859 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11920859 | lifeskim:mentions | umls-concept:C0289174 | lld:lifeskim |
| pubmed-article:11920859 | lifeskim:mentions | umls-concept:C0150632 | lld:lifeskim |
| pubmed-article:11920859 | lifeskim:mentions | umls-concept:C0221099 | lld:lifeskim |
| pubmed-article:11920859 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:11920859 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:11920859 | pubmed:dateCreated | 2002-3-28 | lld:pubmed |
| pubmed-article:11920859 | pubmed:abstractText | Patients homozygous for the C allele of the T102C serotonin (5-HT) 2a receptor polymorphism have shown increased suicidal ideation or behavior in some reports, but not in others. We conducted a pilot investigation to determine whether this polymorphism might relate more specifically to a dimension of impaired impulse control, which may underlie only a portion of suicides. Rates of commission (impulsive) errors in a variant of the Continuous Performance Test (CPT) were compared across the genotypes of the T102C polymorphism in adults recruited from the community. The 102C/102C genotype was jointly associated with a greater incidence of past mood disorder or substance-use disorder, as well as significantly more commission errors compared to the 102T/102C and 102C/102C genotypes. These preliminary data suggest that the T102C 5-HT2a receptor polymorphism may be a marker for impaired behavior control-perhaps in the context of psychiatric disorder history. | lld:pubmed |
| pubmed-article:11920859 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11920859 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11920859 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11920859 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11920859 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11920859 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11920859 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:11920859 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11920859 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:11920859 | pubmed:issn | 0148-7299 | lld:pubmed |
| pubmed-article:11920859 | pubmed:author | pubmed-author:HanisCraig... | lld:pubmed |
| pubmed-article:11920859 | pubmed:author | pubmed-author:DoughertyDona... | lld:pubmed |
| pubmed-article:11920859 | pubmed:author | pubmed-author:SwannAlan CAC | lld:pubmed |
| pubmed-article:11920859 | pubmed:author | pubmed-author:BjorkJames... | lld:pubmed |
| pubmed-article:11920859 | pubmed:author | pubmed-author:MoellerF... | lld:pubmed |
| pubmed-article:11920859 | pubmed:author | pubmed-author:MachadoMirta... | lld:pubmed |
| pubmed-article:11920859 | pubmed:copyrightInfo | Copyright 2002 Wiley-Liss, Inc. | lld:pubmed |
| pubmed-article:11920859 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11920859 | pubmed:day | 8 | lld:pubmed |
| pubmed-article:11920859 | pubmed:volume | 114 | lld:pubmed |
| pubmed-article:11920859 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11920859 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11920859 | pubmed:pagination | 336-9 | lld:pubmed |
| pubmed-article:11920859 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:11920859 | pubmed:meshHeading | pubmed-meshheading:11920859... | lld:pubmed |
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| pubmed-article:11920859 | pubmed:meshHeading | pubmed-meshheading:11920859... | lld:pubmed |
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| pubmed-article:11920859 | pubmed:meshHeading | pubmed-meshheading:11920859... | lld:pubmed |
| pubmed-article:11920859 | pubmed:meshHeading | pubmed-meshheading:11920859... | lld:pubmed |
| pubmed-article:11920859 | pubmed:meshHeading | pubmed-meshheading:11920859... | lld:pubmed |
| pubmed-article:11920859 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:11920859 | pubmed:articleTitle | Serotonin 2a receptor T102C polymorphism and impaired impulse control. | lld:pubmed |
| pubmed-article:11920859 | pubmed:affiliation | Neurobehavioral Research Laboratory and Clinic, Department of Psychiatry and Behavioral Sciences, University of Texas-Houston Medical School, Houston, Texas 77030, USA. | lld:pubmed |
| pubmed-article:11920859 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11920859 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:11920859 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:3356 | entrezgene:pubmed | pubmed-article:11920859 | lld:entrezgene |
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