| pubmed-article:11895222 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11895222 | lifeskim:mentions | umls-concept:C0019932 | lld:lifeskim |
| pubmed-article:11895222 | lifeskim:mentions | umls-concept:C0057277 | lld:lifeskim |
| pubmed-article:11895222 | lifeskim:mentions | umls-concept:C1442792 | lld:lifeskim |
| pubmed-article:11895222 | lifeskim:mentions | umls-concept:C0117002 | lld:lifeskim |
| pubmed-article:11895222 | lifeskim:mentions | umls-concept:C0205216 | lld:lifeskim |
| pubmed-article:11895222 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:11895222 | lifeskim:mentions | umls-concept:C1707455 | lld:lifeskim |
| pubmed-article:11895222 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:11895222 | pubmed:dateCreated | 2002-3-15 | lld:pubmed |
| pubmed-article:11895222 | pubmed:abstractText | Dehydroepiandrosterone (DHEA) and DHEA-sulphate (S) have been suggested to play protective roles in many pathological states, some of which are observed in hyperthyroidism. If DHEA and DHEA-S levels change in hyperthyroidism, they might participate as a possible causative link with such pathophysiological changes in hyperthyroidism. However, the CRH-ACTH-DHEA system in hyperthyroidism has not been clearly defined. We examined plasma levels of DHEA and DHEA-S together with ACTH and cortisol in both hyperthyroid (Hyper) and euthyroid states (Eu). | lld:pubmed |
| pubmed-article:11895222 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11895222 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11895222 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11895222 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:11895222 | pubmed:issn | 0300-0664 | lld:pubmed |
| pubmed-article:11895222 | pubmed:author | pubmed-author:HayashiMM | lld:pubmed |
| pubmed-article:11895222 | pubmed:author | pubmed-author:YasudaKK | lld:pubmed |
| pubmed-article:11895222 | pubmed:author | pubmed-author:MuraeHH | lld:pubmed |
| pubmed-article:11895222 | pubmed:author | pubmed-author:AkanDD | lld:pubmed |
| pubmed-article:11895222 | pubmed:author | pubmed-author:KokuboYY | lld:pubmed |
| pubmed-article:11895222 | pubmed:author | pubmed-author:YamakitaNN | lld:pubmed |
| pubmed-article:11895222 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11895222 | pubmed:volume | 55 | lld:pubmed |
| pubmed-article:11895222 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11895222 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11895222 | pubmed:pagination | 797-803 | lld:pubmed |
| pubmed-article:11895222 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
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| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:meshHeading | pubmed-meshheading:11895222... | lld:pubmed |
| pubmed-article:11895222 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11895222 | pubmed:articleTitle | Dehydroepiandrosterone sulphate is increased and dehydroepiandrosterone-response to corticotrophin-releasing hormone is decreased in the hyperthyroid state compared with the euthyroid state. | lld:pubmed |
| pubmed-article:11895222 | pubmed:affiliation | Department of Internal Medicine, Matsunami General Hospital, Kasamatsu, Gifu-Prefecture, Japan. nyamakita@matsunami-hsp.or.jp | lld:pubmed |
| pubmed-article:11895222 | pubmed:publicationType | Journal Article | lld:pubmed |
