pubmed-article:11877351 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11877351 | lifeskim:mentions | umls-concept:C0004153 | lld:lifeskim |
pubmed-article:11877351 | lifeskim:mentions | umls-concept:C1257792 | lld:lifeskim |
pubmed-article:11877351 | lifeskim:mentions | umls-concept:C0009226 | lld:lifeskim |
pubmed-article:11877351 | lifeskim:mentions | umls-concept:C0019878 | lld:lifeskim |
pubmed-article:11877351 | lifeskim:mentions | umls-concept:C0030016 | lld:lifeskim |
pubmed-article:11877351 | lifeskim:mentions | umls-concept:C1527148 | lld:lifeskim |
pubmed-article:11877351 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:11877351 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
pubmed-article:11877351 | pubmed:issue | 9 | lld:pubmed |
pubmed-article:11877351 | pubmed:dateCreated | 2002-3-5 | lld:pubmed |
pubmed-article:11877351 | pubmed:abstractText | It has been established that hyperhomocyst(e)inemia (HHCy) is an independent and graded risk factor for atherosclerosis, although the molecular link to the atherosclerotic process remains obscure. | lld:pubmed |
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pubmed-article:11877351 | pubmed:language | eng | lld:pubmed |
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pubmed-article:11877351 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:11877351 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:11877351 | pubmed:month | Mar | lld:pubmed |
pubmed-article:11877351 | pubmed:issn | 1524-4539 | lld:pubmed |
pubmed-article:11877351 | pubmed:author | pubmed-author:LiHongH | lld:pubmed |
pubmed-article:11877351 | pubmed:author | pubmed-author:GoligorskyMic... | lld:pubmed |
pubmed-article:11877351 | pubmed:author | pubmed-author:LewisAvalynA | lld:pubmed |
pubmed-article:11877351 | pubmed:author | pubmed-author:BrodskySergey... | lld:pubmed |
pubmed-article:11877351 | pubmed:author | pubmed-author:RiegerRobertR | lld:pubmed |
pubmed-article:11877351 | pubmed:author | pubmed-author:IdenCharlesC | lld:pubmed |
pubmed-article:11877351 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:11877351 | pubmed:day | 5 | lld:pubmed |
pubmed-article:11877351 | pubmed:volume | 105 | lld:pubmed |
pubmed-article:11877351 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:11877351 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:11877351 | pubmed:pagination | 1037-43 | lld:pubmed |
pubmed-article:11877351 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:11877351 | pubmed:year | 2002 | lld:pubmed |
pubmed-article:11877351 | pubmed:articleTitle | Homocysteine induces 3-hydroxy-3-methylglutaryl coenzyme a reductase in vascular endothelial cells: a mechanism for development of atherosclerosis? | lld:pubmed |
pubmed-article:11877351 | pubmed:affiliation | Department of Medicine, State University of New York, Stony Brook, NY 11794-8152, USA. hongli888@yahoo.com | lld:pubmed |
pubmed-article:11877351 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:11877351 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:11877351 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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