| pubmed-article:11815431 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11815431 | lifeskim:mentions | umls-concept:C1706515 | lld:lifeskim |
| pubmed-article:11815431 | lifeskim:mentions | umls-concept:C0162871 | lld:lifeskim |
| pubmed-article:11815431 | lifeskim:mentions | umls-concept:C1514559 | lld:lifeskim |
| pubmed-article:11815431 | lifeskim:mentions | umls-concept:C0210492 | lld:lifeskim |
| pubmed-article:11815431 | lifeskim:mentions | umls-concept:C1704640 | lld:lifeskim |
| pubmed-article:11815431 | lifeskim:mentions | umls-concept:C0677535 | lld:lifeskim |
| pubmed-article:11815431 | lifeskim:mentions | umls-concept:C0331858 | lld:lifeskim |
| pubmed-article:11815431 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:11815431 | pubmed:dateCreated | 2002-1-29 | lld:pubmed |
| pubmed-article:11815431 | pubmed:abstractText | Atherosclerosis is implicated in the pathogenesis of abdominal aortic aneurysm (AAA) but more often causes aortic occlusive disease (AOD). The matrix metalloproteinases (MMPs) degrade extracellular matrix and may play a central role in the pathogenesis of AAA. The aim of this study was to examine differences in the patterns of MMP and MMP inhibitor expression between AAA and AOD. | lld:pubmed |
| pubmed-article:11815431 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11815431 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11815431 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11815431 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:11815431 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11815431 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:11815431 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:11815431 | pubmed:author | pubmed-author:CarrellTom... | lld:pubmed |
| pubmed-article:11815431 | pubmed:author | pubmed-author:BurnandKevin... | lld:pubmed |
| pubmed-article:11815431 | pubmed:author | pubmed-author:WellsGraham... | lld:pubmed |
| pubmed-article:11815431 | pubmed:author | pubmed-author:ClementsJohn... | lld:pubmed |
| pubmed-article:11815431 | pubmed:author | pubmed-author:SmithAlbertoA | lld:pubmed |
| pubmed-article:11815431 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:11815431 | pubmed:day | 29 | lld:pubmed |
| pubmed-article:11815431 | pubmed:volume | 105 | lld:pubmed |
| pubmed-article:11815431 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11815431 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11815431 | pubmed:pagination | 477-82 | lld:pubmed |
| pubmed-article:11815431 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:11815431 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:11815431 | pubmed:articleTitle | Stromelysin-1 (matrix metalloproteinase-3) and tissue inhibitor of metalloproteinase-3 are overexpressed in the wall of abdominal aortic aneurysms. | lld:pubmed |
| pubmed-article:11815431 | pubmed:affiliation | Academic Department of Surgery, Kings College, London, UK. | lld:pubmed |
| pubmed-article:11815431 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11815431 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:11815431 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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