| pubmed-article:11703202 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11703202 | lifeskim:mentions | umls-concept:C0009450 | lld:lifeskim |
| pubmed-article:11703202 | lifeskim:mentions | umls-concept:C0032914 | lld:lifeskim |
| pubmed-article:11703202 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:11703202 | pubmed:dateCreated | 2001-11-12 | lld:pubmed |
| pubmed-article:11703202 | pubmed:abstractText | Studies have suggested a strong paternal factor in the etiology of preeclampsia. If preeclampsia is caused by an infectious agent transmitted by the woman's partner, seronegative women who may experience primary infection in pregnancy should be at increased risk of preeclampsia as compared to previously infected women. The aim of this study was to assess the impact of being seronegative for some viruses transmitted by close contact on the risk of developing preeclampsia. | lld:pubmed |
| pubmed-article:11703202 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11703202 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11703202 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11703202 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11703202 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11703202 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11703202 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:11703202 | pubmed:issn | 0001-6349 | lld:pubmed |
| pubmed-article:11703202 | pubmed:author | pubmed-author:JeanssonSS | lld:pubmed |
| pubmed-article:11703202 | pubmed:author | pubmed-author:EskildAA | lld:pubmed |
| pubmed-article:11703202 | pubmed:author | pubmed-author:BrukA ZAZ | lld:pubmed |
| pubmed-article:11703202 | pubmed:author | pubmed-author:JenumP APA | lld:pubmed |
| pubmed-article:11703202 | pubmed:author | pubmed-author:TrogstadL ILI | lld:pubmed |
| pubmed-article:11703202 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11703202 | pubmed:volume | 80 | lld:pubmed |
| pubmed-article:11703202 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11703202 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11703202 | pubmed:pagination | 1036-8 | lld:pubmed |
| pubmed-article:11703202 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:11703202 | pubmed:meshHeading | pubmed-meshheading:11703202... | lld:pubmed |
| pubmed-article:11703202 | pubmed:meshHeading | pubmed-meshheading:11703202... | lld:pubmed |
| pubmed-article:11703202 | pubmed:meshHeading | pubmed-meshheading:11703202... | lld:pubmed |
| pubmed-article:11703202 | pubmed:meshHeading | pubmed-meshheading:11703202... | lld:pubmed |
| pubmed-article:11703202 | pubmed:meshHeading | pubmed-meshheading:11703202... | lld:pubmed |
| pubmed-article:11703202 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11703202 | pubmed:articleTitle | Is preeclampsia an infectious disease? | lld:pubmed |
| pubmed-article:11703202 | pubmed:affiliation | Department of Population Health, National Institute of Public Health, P.O. Box 4404 Nydalen, 0403 Oslo, Norway. lill.trogstad@folkehelsa.no | lld:pubmed |
| pubmed-article:11703202 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11703202 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:11703202 | lld:pubmed |
