| pubmed-article:11698122 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11698122 | lifeskim:mentions | umls-concept:C0039195 | lld:lifeskim |
| pubmed-article:11698122 | lifeskim:mentions | umls-concept:C0002874 | lld:lifeskim |
| pubmed-article:11698122 | lifeskim:mentions | umls-concept:C0026985 | lld:lifeskim |
| pubmed-article:11698122 | lifeskim:mentions | umls-concept:C0031437 | lld:lifeskim |
| pubmed-article:11698122 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:11698122 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:11698122 | pubmed:dateCreated | 2001-11-7 | lld:pubmed |
| pubmed-article:11698122 | pubmed:abstractText | We hypothesized that an active autoimmune process in aplastic anemia (AA) corresponds to the expansion of cytotoxic lymphocytes (CTLs) displaying mature effector phenotype. We determined whether the numbers of effector CTLs in blood of patients with bone marrow failure syndromes are elevated and correlate with the disease activity and responsiveness to immunosuppression. | lld:pubmed |
| pubmed-article:11698122 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11698122 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11698122 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11698122 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11698122 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:11698122 | pubmed:issn | 0301-472X | lld:pubmed |
| pubmed-article:11698122 | pubmed:author | pubmed-author:KookHH | lld:pubmed |
| pubmed-article:11698122 | pubmed:author | pubmed-author:YoungN SNS | lld:pubmed |
| pubmed-article:11698122 | pubmed:author | pubmed-author:KirbyMM | lld:pubmed |
| pubmed-article:11698122 | pubmed:author | pubmed-author:ZengWW | lld:pubmed |
| pubmed-article:11698122 | pubmed:author | pubmed-author:MaciejewskiJ... | lld:pubmed |
| pubmed-article:11698122 | pubmed:author | pubmed-author:GuibinCC | lld:pubmed |
| pubmed-article:11698122 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11698122 | pubmed:volume | 29 | lld:pubmed |
| pubmed-article:11698122 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11698122 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11698122 | pubmed:pagination | 1270-7 | lld:pubmed |
| pubmed-article:11698122 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:11698122 | pubmed:meshHeading | pubmed-meshheading:11698122... | lld:pubmed |
| pubmed-article:11698122 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11698122 | pubmed:articleTitle | Increased cytotoxic T cells with effector phenotype in aplastic anemia and myelodysplasia. | lld:pubmed |
| pubmed-article:11698122 | pubmed:affiliation | Hematology Branch, National Heart, Lung and Blood Institute, National Institutes of Health, Bethesda, MD 20892, USA. | lld:pubmed |
| pubmed-article:11698122 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11698122 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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