pubmed-article:11684016 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C0031715 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C0007620 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C0010656 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C0205245 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C1419744 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C1419742 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C1419743 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C0179400 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C1706214 | lld:lifeskim |
pubmed-article:11684016 | lifeskim:mentions | umls-concept:C1511545 | lld:lifeskim |
pubmed-article:11684016 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:11684016 | pubmed:dateCreated | 2001-10-30 | lld:pubmed |
pubmed-article:11684016 | pubmed:abstractText | Upon activation by liver injury, hepatic stellate cells produce excessive fibrous tissue leading to cirrhosis. The hepatotoxin CCl(4) induced activation of RSK, phosphorylation of C/EBPbeta on Thr(217), and proliferation of stellate cells in normal mice, but caused apoptosis of these cells in C/EBPbeta-/- or C/EBPbeta-Ala(217) (a dominant-negative nonphosphorylatable mutant) transgenic mice. Both C/EBPbeta-PThr(217) and the phosphorylation mimic C/EBPbeta-Glu(217), but not C/EBPbeta-Ala(217), were associated with procaspases 1 and 8 in vivo and in vitro and inhibited their activation. Our data suggest that C/EBPbeta phosphorylation on Thr(217) creates a functional XEXD caspase substrate/inhibitor box (K-Phospho-T(217)VD) that is mimicked by C/EBPbeta-Glu(217) (KE(217)VD). C/EBPbeta-/- and C/EBPbeta-Ala(217) stellate cells were rescued from apoptosis by the cell permeant KE(217)VD tetrapeptide or C/EBPbeta-Glu(217). | lld:pubmed |
pubmed-article:11684016 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11684016 | pubmed:language | eng | lld:pubmed |
pubmed-article:11684016 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11684016 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:11684016 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:11684016 | pubmed:month | Oct | lld:pubmed |
pubmed-article:11684016 | pubmed:issn | 1097-2765 | lld:pubmed |
pubmed-article:11684016 | pubmed:author | pubmed-author:PoliVV | lld:pubmed |
pubmed-article:11684016 | pubmed:author | pubmed-author:HunterTT | lld:pubmed |
pubmed-article:11684016 | pubmed:author | pubmed-author:BuckMM | lld:pubmed |
pubmed-article:11684016 | pubmed:author | pubmed-author:ChojkierMM | lld:pubmed |
pubmed-article:11684016 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:11684016 | pubmed:volume | 8 | lld:pubmed |
pubmed-article:11684016 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:11684016 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:11684016 | pubmed:pagination | 807-16 | lld:pubmed |
pubmed-article:11684016 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:11684016 | pubmed:year | 2001 | lld:pubmed |
pubmed-article:11684016 | pubmed:articleTitle | C/EBPbeta phosphorylation by RSK creates a functional XEXD caspase inhibitory box critical for cell survival. | lld:pubmed |
pubmed-article:11684016 | pubmed:affiliation | Molecular Biology and Virology Laboratory, The Salk Institute for Biological Studies, La Jolla, CA 92037, USA. | lld:pubmed |
pubmed-article:11684016 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:11684016 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:11684016 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
pubmed-article:11684016 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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