| pubmed-article:11676556 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11676556 | lifeskim:mentions | umls-concept:C0009450 | lld:lifeskim |
| pubmed-article:11676556 | lifeskim:mentions | umls-concept:C0599779 | lld:lifeskim |
| pubmed-article:11676556 | lifeskim:mentions | umls-concept:C0005411 | lld:lifeskim |
| pubmed-article:11676556 | lifeskim:mentions | umls-concept:C0061928 | lld:lifeskim |
| pubmed-article:11676556 | lifeskim:mentions | umls-concept:C1550587 | lld:lifeskim |
| pubmed-article:11676556 | lifeskim:mentions | umls-concept:C0205470 | lld:lifeskim |
| pubmed-article:11676556 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:11676556 | pubmed:dateCreated | 2001-10-25 | lld:pubmed |
| pubmed-article:11676556 | pubmed:abstractText | Extrahepatic biliary atresia (EHBA), the etiology of which still remains unclear, occurs exclusively in newborns and has recently been simulated in an animal model. It is possible to trigger an EHBA corresponding to the human disease by means of intraperitoneal infection of newborn Balb/c mice with rhesus rotavirus (RRV). The aim of the present study was to determine the conditions and circumstances for inducing biliary atresia in this model focusing on first-line immunological aspects. | lld:pubmed |
| pubmed-article:11676556 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11676556 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11676556 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11676556 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11676556 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:11676556 | pubmed:issn | 0022-4804 | lld:pubmed |
| pubmed-article:11676556 | pubmed:author | pubmed-author:VerhagenWW | lld:pubmed |
| pubmed-article:11676556 | pubmed:author | pubmed-author:PetersenCC | lld:pubmed |
| pubmed-article:11676556 | pubmed:author | pubmed-author:LeonhardtJJ | lld:pubmed |
| pubmed-article:11676556 | pubmed:author | pubmed-author:SzavayPP | lld:pubmed |
| pubmed-article:11676556 | pubmed:author | pubmed-author:Czech-Schmidt... | lld:pubmed |
| pubmed-article:11676556 | pubmed:copyrightInfo | Copyright 2001 Academic Press. | lld:pubmed |
| pubmed-article:11676556 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11676556 | pubmed:volume | 101 | lld:pubmed |
| pubmed-article:11676556 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11676556 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11676556 | pubmed:pagination | 62-7 | lld:pubmed |
| pubmed-article:11676556 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:11676556 | pubmed:meshHeading | pubmed-meshheading:11676556... | lld:pubmed |
| pubmed-article:11676556 | pubmed:meshHeading | pubmed-meshheading:11676556... | lld:pubmed |
| pubmed-article:11676556 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11676556 | pubmed:articleTitle | Immunological gap in the infectious animal model for biliary atresia. | lld:pubmed |
| pubmed-article:11676556 | pubmed:affiliation | Department of Pediatric Surgery, Medical School Hannover, Carl-Neuberg-Strasse 1, Hannover, 30625, Germany. | lld:pubmed |
| pubmed-article:11676556 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11676556 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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