| pubmed-article:11665973 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11665973 | lifeskim:mentions | umls-concept:C0035648 | lld:lifeskim |
| pubmed-article:11665973 | lifeskim:mentions | umls-concept:C0024141 | lld:lifeskim |
| pubmed-article:11665973 | lifeskim:mentions | umls-concept:C1849618 | lld:lifeskim |
| pubmed-article:11665973 | lifeskim:mentions | umls-concept:C1548559 | lld:lifeskim |
| pubmed-article:11665973 | lifeskim:mentions | umls-concept:C1551359 | lld:lifeskim |
| pubmed-article:11665973 | lifeskim:mentions | umls-concept:C0443324 | lld:lifeskim |
| pubmed-article:11665973 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:11665973 | pubmed:dateCreated | 2001-10-22 | lld:pubmed |
| pubmed-article:11665973 | pubmed:abstractText | The frequency of coronary heart disease (CHD) and stroke are increased in systemic lupus erythematosus (SLE), but the extent of the increase is uncertain. We sought to determine to what extent the increase could not be explained by common risk factors. | lld:pubmed |
| pubmed-article:11665973 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11665973 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11665973 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11665973 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:11665973 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11665973 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:11665973 | pubmed:issn | 0004-3591 | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:LUMM | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:ClarkeA EAE | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:EsdaileJ MJM | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:AbrahamowiczM... | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:GroverS ASA | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:SenécalJ LJL | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:CôteRR | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:FortinP RPR | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:du BergerRR | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:GrodzickyTT | lld:pubmed |
| pubmed-article:11665973 | pubmed:author | pubmed-author:PanaritisCC | lld:pubmed |
| pubmed-article:11665973 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11665973 | pubmed:volume | 44 | lld:pubmed |
| pubmed-article:11665973 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11665973 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11665973 | pubmed:pagination | 2331-7 | lld:pubmed |
| pubmed-article:11665973 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:11665973 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11665973 | pubmed:articleTitle | Traditional Framingham risk factors fail to fully account for accelerated atherosclerosis in systemic lupus erythematosus. | lld:pubmed |
| pubmed-article:11665973 | pubmed:affiliation | University of British Columbia, Vancouver, Canada. jesdaile@bc.arthritis.ca | lld:pubmed |
| pubmed-article:11665973 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11665973 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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