| pubmed-article:11597326 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11597326 | lifeskim:mentions | umls-concept:C0346153 | lld:lifeskim |
| pubmed-article:11597326 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:11597326 | lifeskim:mentions | umls-concept:C0021920 | lld:lifeskim |
| pubmed-article:11597326 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
| pubmed-article:11597326 | lifeskim:mentions | umls-concept:C0079419 | lld:lifeskim |
| pubmed-article:11597326 | lifeskim:mentions | umls-concept:C0004340 | lld:lifeskim |
| pubmed-article:11597326 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:11597326 | pubmed:dateCreated | 2001-10-12 | lld:pubmed |
| pubmed-article:11597326 | pubmed:abstractText | Mutations in BRCA1 and BRCA2 account for approximately 50% of breast cancer families with more than four affected cases, whereas exonic mutations in p53, PTEN, CHK2 and ATM may account for a very small proportion. It was recently reported that an intronic variant of p53--G13964C--occurred in three out of 42 (7.1%) 'hereditary' breast cancer patients, but not in any of 171 'sporadic' breast cancer control individuals (P = 0.0003). If this relatively frequent occurrence of G13964C in familial breast cancer and absence in control individuals were confirmed, then this would suggest that the G13964C variant plays a role in breast cancer susceptibility. | lld:pubmed |
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| pubmed-article:11597326 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11597326 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11597326 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11597326 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11597326 | pubmed:issn | 1465-5411 | lld:pubmed |
| pubmed-article:11597326 | pubmed:author | pubmed-author:MarshAA | lld:pubmed |
| pubmed-article:11597326 | pubmed:author | pubmed-author:SambrookJ FJF | lld:pubmed |
| pubmed-article:11597326 | pubmed:author | pubmed-author:HopperJ LJL | lld:pubmed |
| pubmed-article:11597326 | pubmed:author | pubmed-author:ThorneHH | lld:pubmed |
| pubmed-article:11597326 | pubmed:author | pubmed-author:MannG JGJ | lld:pubmed |
| pubmed-article:11597326 | pubmed:author | pubmed-author:TurnerB CBC | lld:pubmed |
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| pubmed-article:11597326 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11597326 | pubmed:volume | 3 | lld:pubmed |
| pubmed-article:11597326 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11597326 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11597326 | pubmed:pagination | 346-9 | lld:pubmed |
| pubmed-article:11597326 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:11597326 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11597326 | pubmed:articleTitle | The intronic G13964C variant in p53 is not a high-risk mutation in familial breast cancer in Australia. | lld:pubmed |
| pubmed-article:11597326 | pubmed:affiliation | Queensland Institute of Medical Research, Brisbane, Queensland, Australia. | lld:pubmed |
| pubmed-article:11597326 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11597326 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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