pubmed-article:11564471 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11564471 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:11564471 | lifeskim:mentions | umls-concept:C0678804 | lld:lifeskim |
pubmed-article:11564471 | lifeskim:mentions | umls-concept:C0240502 | lld:lifeskim |
pubmed-article:11564471 | lifeskim:mentions | umls-concept:C0205949 | lld:lifeskim |
pubmed-article:11564471 | lifeskim:mentions | umls-concept:C0001962 | lld:lifeskim |
pubmed-article:11564471 | lifeskim:mentions | umls-concept:C0456057 | lld:lifeskim |
pubmed-article:11564471 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:11564471 | pubmed:issue | 1-2 | lld:pubmed |
pubmed-article:11564471 | pubmed:dateCreated | 2001-9-20 | lld:pubmed |
pubmed-article:11564471 | pubmed:abstractText | Studies of rats have shown that mothers who are subjected to stress during pregnancy are more likely than mothers who are not stressed during pregnancy to have male offspring who exhibit female-typical sexual receptivity postures (lordosis) in the presence of other males following the onset of puberty. More recent animal experiments have indicated that prenatal exposure to alcohol affects the sexual preferences of male offspring in ways that are similar to the effects of prenatal stress. Research with human subjects have thus far yielded inconsistent findings regarding the effects of prenatal stress on male sexual orientation, and no research has yet addressed the possible involvement of prenatal exposure to alcohol or other widely used recreational drugs, such as nicotine. | lld:pubmed |
pubmed-article:11564471 | pubmed:language | eng | lld:pubmed |
pubmed-article:11564471 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11564471 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:11564471 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11564471 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11564471 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11564471 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11564471 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:11564471 | pubmed:issn | 0031-9384 | lld:pubmed |
pubmed-article:11564471 | pubmed:author | pubmed-author:EllisLL | lld:pubmed |
pubmed-article:11564471 | pubmed:author | pubmed-author:Cole-HardingS... | lld:pubmed |
pubmed-article:11564471 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:11564471 | pubmed:volume | 74 | lld:pubmed |
pubmed-article:11564471 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:11564471 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:11564471 | pubmed:pagination | 213-26 | lld:pubmed |
pubmed-article:11564471 | pubmed:dateRevised | 2004-11-17 | lld:pubmed |
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pubmed-article:11564471 | pubmed:articleTitle | The effects of prenatal stress, and of prenatal alcohol and nicotine exposure, on human sexual orientation. | lld:pubmed |
pubmed-article:11564471 | pubmed:affiliation | Minot State University, 58707, Minot, ND, USA. ellis@minotstateu.edu | lld:pubmed |
pubmed-article:11564471 | pubmed:publicationType | Journal Article | lld:pubmed |
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